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Merck
CN

Alleviation by GABA

Neurochemical research (2017-06-14)
Toshihiko Kinjo, Yoshino Ashida, Hiroshi Higashi, Satoshi Sugimura, Miho Washida, Hiroki Niihara, Kiyokazu Ogita, Yukio Yoneda, Nobuyuki Kuramoto
摘要

Mitochondrial permeability transition pore (PTP) is supposed to at least in part participate in molecular mechanisms underlying the neurotoxicity seen after overactivation of N-methyl-D-aspartate (NMDA) receptor (NMDAR) in neurons. In this study, we have evaluated whether activation of GABA

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Sigma-Aldrich
N-甲基- D -天冬氨酸, ≥98% (TLC), solid
Sigma-Aldrich
R (+)-巴氯芬 盐酸盐, solid
Sigma-Aldrich
3,3′-二丙基硫杂羰花青碘化物, 98%