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安全信息

SAB5201086

Sigma-Aldrich

Monoclonal Anti-Glua1/Glur1 antibody produced in mouse

clone S355-1, purified immunoglobulin

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别名:
Anti-AMPA 1, Anti-GLUH 1, Anti-GluR K1, Anti-Glutamate receptor ionotropic AMPA 1, Anti-HBGR1
UNSPSC代码:
12352203
NACRES:
NA.41

生物来源

mouse

质量水平

偶联物

unconjugated

抗体形式

purified immunoglobulin

抗体产品类型

primary antibodies

克隆

S355-1, monoclonal

形式

buffered aqueous solution

分子量

antigen predicted mol wt 100 kDa

种属反应性

rat, mouse

浓度

1 mg/mL

技术

immunocytochemistry: suitable
immunohistochemistry: suitable
western blot: suitable

同位素/亚型

IgG1

NCBI登记号

UniProt登记号

运输

wet ice

储存温度

−20°C

靶向翻译后修饰

unmodified

基因信息

免疫原

Fusion protein amino acids 1-389 (extracellular N-terminus) of rat GluA1/GluR1

特点和优势

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

外形

PBS pH 7.4, 50% glycerol, 0.1% sodium azide

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

WGK

WGK 1

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

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Dylan J Calame et al.
Neuroscience, 448, 272-286 (2020-09-07)
PRRT2 loss-of-function mutations have been associated with familial paroxysmal kinesigenic dyskinesia (PKD), infantile convulsions and choreoathetosis, and benign familial infantile seizures. Dystonia is the foremost involuntary movement disorder manifest by patients with PKD. Using a lacZ reporter and quantitative reverse-transcriptase
Shinji Matsuda et al.
The Journal of biological chemistry, 297(2), 100949-100949 (2021-07-13)
Long-term potentiation (LTP) and long-term depression (LTD) of excitatory neurotransmission are believed to be the neuronal basis of learning and memory. Both processes are primarily mediated by neuronal activity-induced transport of postsynaptic AMPA-type glutamate receptors (AMPARs). While AMPAR subunits and

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