SAB4501304
Anti-NMDAR2A antibody produced in rabbit
affinity isolated antibody
别名:
GRIN2A, Glutamate [NMDA] receptor subunit ε 1 precursor, N-methyl D-aspartate receptor subtype 2A, N-methyl-D-aspartate receptor subtype 2A, NMDAR2AGlutamate [NMDA] receptor subunit ε 1
产品名称
Anti-NMDAR2A antibody produced in rabbit, affinity isolated antibody
biological source
rabbit
conjugate
unconjugated
antibody form
affinity isolated antibody
antibody product type
primary antibodies
clone
polyclonal
form
buffered aqueous solution
mol wt
antigen 165 kDa
species reactivity
rat, mouse, human
concentration
~1 mg/mL
technique(s)
ELISA: 1:1000
immunofluorescence: 1:100-1:500
immunohistochemistry: 1:50-1:100
NCBI accession no.
UniProt accession no.
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
unmodified
Quality Level
Gene Information
human ... GRIN2A(2903)
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
Features and Benefits
Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.
General description
Anti-NMDAR2A Antibody detects endogenous levels of total NMDAR2A protein.
Immunogen
The antiserum was produced against synthesized peptide derived from human NMDAR2A/B.
Immunogen Range: 1216-1265
Immunogen Range: 1216-1265
Physical form
Rabbit IgG in phosphate buffered saline (without Mg2+ and Ca2+), pH 7.4, 150mM NaCl, 0.02% sodium azide and 50% glycerol.
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存储类别
10 - Combustible liquids
wgk
nwg
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
常规特殊物品
此项目有
Wei-Yi Gong et al.
Frontiers in aging neuroscience, 9, 123-123 (2017-05-30)
Many factors impact cognitive impairment; however, the effects of chronic pain and the mechanisms underlying these effects on cognitive impairment are currently unknown. Here we tested the hypothesis that chronic pain accelerates the transition from normal cognition to mild cognitive
Przemysław Duda et al.
Aging, 10(7), 1682-1697 (2018-07-22)
Aging is believed to be the result of alterations of protein expression and accumulation of changes in biomolecules. Although there are numerous reports demonstrating changes in protein expression in brain during aging, only few of them describe global changes at
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