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安全信息

SAB3500018

Sigma-Aldrich

抗 OTUD4 兔抗

affinity isolated antibody, buffered aqueous solution

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别名:
Anti-DUBA6, Anti-HIN1, Anti-HSHIN1, Anti-OTU domain containing 4
UNSPSC代码:
12352203
NACRES:
NA.41

生物来源

rabbit

质量水平

偶联物

unconjugated

抗体形式

affinity isolated antibody

抗体产品类型

primary antibodies

克隆

polyclonal

形式

buffered aqueous solution

分子量

predicted mol wt 115-123 kDa

种属反应性

mouse, human, rat

技术

immunocytochemistry: suitable
indirect ELISA: suitable
western blot: suitable

NCBI登记号

UniProt登记号

运输

dry ice

储存温度

−20°C

靶向翻译后修饰

unmodified

基因信息

human ... OTUD4(54726)

免疫原

OTUD4 antibody was raised against an 18 amino acid peptide near the carboxy terminus of human OTUD4.

特点和优势

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

目标描述

OTUD4, also known as HIV-1 induced protein HIN-1, is a member of the OTU (ovarian tumor) domain containing cysteine protease superfamily, in which the OUT domain generally confers deubiquitinase activity. At least three isoforms of OTUD4 are known to exist, and the smallest of these isoforms are only expressed in HIV-1-infected cells (provided by RefSeq). This antibody is predicted to not cross-react with other members of the OTUD family.

联系

The action of this antibody can be blocked using blocking peptide SBP3500018.

外形

Supplied at approx. 1 mg/mL in phosphate buffered saline containing 0.02% sodium azide.

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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WGK

WGK 2

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

常规特殊物品

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Muping Di et al.
Journal of experimental & clinical cancer research : CR, 41(1), 328-328 (2022-11-22)
Radioresistance is the primary cause of nasopharyngeal carcinoma (NPC) treatment failure. Previous studies have focused on the deficits in cellular apoptosis as a mechanism for radioresistance; however, additional potential death modes involved in modulating radiosensitivity of NPC have not been

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