推荐产品
生物来源
mouse
质量水平
偶联物
unconjugated
抗体形式
affinity isolated antibody
抗体产品类型
primary antibodies
克隆
GT844, monoclonal
表单
buffered aqueous solution
分子量
27kDa
浓度
1mg/mL
技术
immunofluorescence: 1:100-1:1,000
western blot: 1:500-1:3,000
同位素/亚型
IgG2a
运输
wet ice
储存温度
−20°C
靶向翻译后修饰
unmodified
一般描述
The acceptor mCherry is a monomeric, red fluorescent protein obtained from the stony-coral Discosoma protein DsRED. It is expressed in the cytoplasm, has a high photostability and is unaffected by photobleaching. mCherry acts as a good FRET (fluorescence resonance energy transfer) acceptor. mCherry allows rapid fluorescence detection after a particular gene is expressed.
免疫原
Full length mCherry recombinant protein
应用
Suggested starting dilutions are as follows: WB: 1:500-1:3000. Not yet tested in other applications. Optimal working dilutions should be determined experimentally by the end user.
特点和优势
Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.
其他说明
Purification: Affinity purified by Protein G
外形
Phosphate-buffered saline, no preservative added.
免责声明
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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储存分类代码
12 - Non Combustible Liquids
WGK
WGK 2
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
新产品
In vitro screening test using Leishmania promastigotes stably expressing mCherry protein.
Antimicrobial Agents and Chemotherapy, 58(3), 1825-1828 (2014)
Quantitative FRET Analysis With the E0GFP?mCherry Fluorescent Protein Pair.
Photochemistry and Photobiology, 85(1), 287-297 (2009)
Nature neuroscience, 26(3), 458-469 (2023-01-24)
Poor sleep is associated with the risk of developing chronic pain, but how sleep contributes to pain chronicity remains unclear. Here we show that following peripheral nerve injury, cholinergic neurons in the anterior nucleus basalis (aNB) of the basal forebrain
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