推荐产品
生物来源
synthetic (organic)
质量水平
方案
≥75% (HPLC)
表单
powder
颜色
off-white
溶解性
H2O: 25 mg/mL
运输
dry ice
储存温度
−20°C
SMILES字符串
[Li+].[Li+].[Li+].[Li+].Nc1ncnc2n(cnc12)[C@@H]3O[C@H](COP([O-])(=O)OP([O-])(=O)OP([O-])([O-])=S)[C@@H](O)[C@H]3O
InChI
1S/C10H16N5O12P3S.4Li/c11-8-5-9(13-2-12-8)15(3-14-5)10-7(17)6(16)4(25-10)1-24-28(18,19)26-29(20,21)27-30(22,23)31;;;;/h2-4,6-7,10,16-17H,1H2,(H,18,19)(H,20,21)(H2,11,12,13)(H2,22,23,31);;;;/q;4*+1/p-4/t4-,6-,7-,10-;;;;/m1..../s1
InChI key
DWQFDOIBOYDYKH-KWIZKVQNSA-J
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相关类别
应用
腺苷5′-[γ-硫代]三磷酸腺苷四锂盐已用于处理花粉粒，测定三磷酸腺苷（ATP）或其水解产物的抑制作用。它还用于筛选硫代磷酸酯靶蛋白。
生化/生理作用
腺苷5′-[γ-硫代]三磷酸(ATP-γ-S)是一种广泛使用的非水解ATP类似物，用于研究包括细胞外功能等多种过程中ATP结合ATP位点的影响。ATP- γ-S是一种P2嘌呤能激动剂，能够增强Ca2+-活化 K+通道活性。 ATP- γ-S能够在激酶反应中取代ATP，生成硫代磷酸化蛋白，耐受蛋白磷酸酶，调节泛素偶联物和26S蛋白酶体结合的ATP依赖性步骤。 ATP-γ -S可差分调节Toll样受体4介导的细胞存活和死亡。
其他说明
非水解ATP类似物
储存分类代码
11 - Combustible Solids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
dust mask type N95 (US), Eyeshields, Gloves
从最新的版本中选择一种：
分析证书(COA)
Lot/Batch Number
Andreas Peth et al.
Molecular cell, 40(4), 671-681 (2010-11-26)
Eukaryotic cells target proteins for degradation by the 26S proteasome by attaching a ubiquitin chain. Using a rapid assay, we analyzed the initial binding of ubiquitinated proteins to purified 26S particles as an isolated process at 4°C. Subunits Rpn10 and
S Chrysogelos et al.
The Journal of biological chemistry, 258(20), 12624-12631 (1983-10-25)
recA protein binds to duplex DNA in the presence of Mg2+ and adenosine 5'-O-(3-thiotriphosphate) forming a stiff nucleoprotein filament with a distinct axial repeat which contains 17 +/- 1 base pairs and spans 8-9 nm along the fiber (Di Capua
David M Smith et al.
Cell, 144(4), 526-538 (2011-02-22)
In the eukaryotic 26S proteasome, the 20S particle is regulated by six AAA ATPase subunits and, in archaea, by a homologous ring complex, PAN. To clarify the role of ATP in proteolysis, we studied how nucleotides bind to PAN. Although
Labeling and identification of direct kinase substrates
Carlson SM and White FM
Science Signaling, 5(227), pl3-pl3 (2012)
Kana Harada et al.
Journal of neurochemistry, 116(6), 1138-1147 (2011-01-08)
The survival and death rates of inflammatory cells directly control their number and are substantially associated with the degree of inflammation. Microglia, key players in neuroinflammation, often cause excessive reactions implicated in neurological diseases. However, the mechanisms that determine microglial
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