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Merck
CN
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AG252

Sigma-Aldrich

GAD Peptide

control for AB1511, powder

别名:

GAD

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About This Item

UNSPSC代码:
12352203
eCl@ss:
32160702
NACRES:
NA.77

product name

GAD, control peptide for AB1511,

生物来源

rat

质量水平

检测方案

>80%

形式

powder

制造商/商品名称

Chemicon®

技术

cell based assay: suitable

NCBI登记号

UniProt登记号

运输

dry ice

基因信息

rat ... GAD2(24380)

一般描述

Synthetic peptide corresponding to amino acid residues Cys+ 572-585 of rat GAD65 (Erlander et al., 1991). The amino acid sequence is wholly conserved in rat GAD67, and the peptide is located at the C-terminus in both forms of the enzyme. Sequence: [C]DFLIEEIERLGQDL.

应用

Antibody blocking (AB1511)

Optimal working dilution must be determined by the end user.
Research Category
Neuroscience
Research Sub Category
Neurotransmitters & Receptors

Neuronal & Glial Markers

外形

White lyophilized powder. Soluble in water.

储存及稳定性

Long term storage at -20°C in a sealed container with desiccant for up to 12 months after date of receipt.

法律信息

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

储存分类代码

11 - Combustible Solids

WGK

WGK 1

闪点(°F)

Not applicable

闪点(°C)

Not applicable


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M G Erlander et al.
Neuron, 7(1), 91-100 (1991-07-01)
gamma-Aminobutyric acid (GABA) is the most widely distributed known inhibitory neurotransmitter in the vertebrate brain. GABA also serves regulatory and trophic roles in several other organs, including the pancreas. The brain contains two forms of the GABA synthetic enzyme glutamate
Louis-Etienne Lorenzo et al.
Nature communications, 11(1), 869-869 (2020-02-15)
Spinal disinhibition has been hypothesized to underlie pain hypersensitivity in neuropathic pain. Apparently contradictory mechanisms have been reported, raising questions on the best target to produce analgesia. Here, we show that nerve injury is associated with a reduction in the

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