产品名称
Goat Anti-Human IgE, ε-Chain Specific, liquid, Calbiochem®
biological source
goat
antibody form
diluted serum
antibody product type
secondary antibodies
clone
polyclonal
form
liquid
contains
≤0.1% sodium azide as preservative
species reactivity
human
manufacturer/tradename
Calbiochem®
storage condition
OK to freeze
avoid repeated freeze/thaw cycles
dilution
(Immunoelectrophoresis )
isotype
IgG
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
unmodified
Quality Level
Application
Immunoelectrophoresis (see comments)
Disclaimer
Toxicity: Standard Handling (A)
General description
Goat polyclonal antibody supplied as sterile-filtered, defibrinated, delipidized, and adsorbed serum. Recognizes human IgE ε-chain.
This Goat Anti-Human IgE, ε-Chain Specific is validated for use in Immunoelectrophoresis for the detection of Human IgE, ε-Chain Specific.
Immunogen
Human
Other Notes
Monospecific for human IgE ε-chain as demonstrated by a single arc when tested against IgE-elevated serum. Variables associated with assay conditions will dictate the proper working dilution.
Packaging
Please refer to vial label for lot-specific concentration.
Physical form
Serum diluted in 500 mM NaCl, 50 mM Tris-HCl, pH 7.5.
Preparation Note
Following initial thaw, aliquot and freeze (-20°C).
Legal Information
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
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存储类别
12 - Non Combustible Liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
S Sievers et al.
Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology, 47(8), 1057-1068 (2017-03-28)
Microarray-based component-resolved diagnostics (CRD) has become an accepted tool to detect allergen-specific IgE sensitization towards hundreds of allergens in parallel from one drop of serum. Nevertheless, specificity and sensitivity as well as a simultaneous detection of allergen-specific IgG4 , as
Henry R Maun et al.
Cell, 179(2), 417-431 (2019-10-05)
Severe asthma patients with low type 2 inflammation derive less clinical benefit from therapies targeting type 2 cytokines and represent an unmet need. We show that mast cell tryptase is elevated in severe asthma patients independent of type 2 biomarker
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