E-041
乙醇-150
150 mg/dL in H2O, ampule of 10 × 1.2 mL, certified reference material, Cerilliant®
别名:
Ethanol 溶液, 乙基醇
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关于此项目
线性分子式:
C2H5OH
化学文摘社编号:
分子量:
46.07
UNSPSC Code:
41116107
NACRES:
NA.24
PubChem Substance ID:
MDL number:
Beilstein/REAXYS Number:
1718733
InChI
1S/C2H6O/c1-2-3/h3H,2H2,1H3
SMILES string
CCO
InChI key
LFQSCWFLJHTTHZ-UHFFFAOYSA-N
grade
certified reference material
form
liquid
feature
Snap-N-Spike®/Snap-N-Shoot®
packaging
ampule of 10 × 1.2 mL
manufacturer/tradename
Cerilliant®
concentration
150 mg/dL in H2O
technique(s)
gas chromatography (GC): suitable, liquid chromatography (LC): suitable
application(s)
forensics and toxicology
format
single component solution
storage temp.
2-8°C
Quality Level
General description
适用于校准乙醇检测方法的NIST可追溯乙醇标准品试剂盒。这些认证参考物质广泛应用于法医分析和临床毒理学，用于血液中乙醇含量的测定。
Legal Information
CERILLIANT is a registered trademark of Merck KGaA, Darmstadt, Germany
Snap-N-Shoot is a registered trademark of Cerilliant Corporation
Snap-N-Spike is a registered trademark of Merck KGaA, Darmstadt, Germany
存储类别
10 - Combustible liquids
wgk
nwg
flash_point_f
Not applicable
flash_point_c
Not applicable
S Enocksson et al.
The Journal of clinical investigation, 95(5), 2239-2245 (1995-05-01)
Although it is well established in several mammalian species that beta 3-adrenoceptors play a major role in regulating lipolysis and thermogenesis in adipose tissue, the functional existence and role of this receptor subtype in man has been controversial. We investigated
Kazue Hashimoto-Torii et al.
Neuron, 82(3), 560-572 (2014-04-15)
Prenatal exposure of the developing brain to various environmental challenges increases susceptibility to late onset of neuropsychiatric dysfunction; still, the underlying mechanisms remain obscure. Here we show that exposure of embryos to a variety of environmental factors such as alcohol
Irene Zelner et al.
Drug metabolism reviews, 45(3), 277-299 (2013-05-30)
The ability to undergo non-oxidative metabolism from ethanol to fatty acid ethyl esters (FAEEs) varies greatly among tissues and organs. To gain a greater understanding of non-oxidative ethanol metabolism to FAEE, we aimed to collect all published data on FAEE
Elhaseen Elamin et al.
American journal of physiology. Gastrointestinal and liver physiology, 306(8), G677-G685 (2014-02-22)
Evidence indicates that ethanol-induced intestinal barrier dysfunction and subsequent endotoxemia plays a key role in the pathogenesis of alcoholic liver disease. Recently, it has been demonstrated that ethanol induces RhoA kinase activation in intestinal epithelium, thereby disrupting barrier integrity. In
Carlo Angelici et al.
ChemSusChem, 6(9), 1595-1614 (2013-05-25)
The development of new and improved processes for the synthesis of bio-based chemicals is one of the scientific challenges of our time. These new discoveries are not only important from an environmental point of view, but also represent an important
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