跳转至内容
Merck
CN
  • KCa3.1 Inhibition Switches the Astrocyte Phenotype during Astrogliosis Associated with Ischemic Stroke Via Endoplasmic Reticulum Stress and MAPK Signaling Pathways.

KCa3.1 Inhibition Switches the Astrocyte Phenotype during Astrogliosis Associated with Ischemic Stroke Via Endoplasmic Reticulum Stress and MAPK Signaling Pathways.

Frontiers in cellular neuroscience (2017-10-28)
Zhihua Yu, Mengni Yi, Tianjiao Wei, Xiaoling Gao, Hongzhuan Chen
摘要

Ischemic stroke is a devastating neurological disease that can initiate a phenotype switch in astrocytes. Reactive astrogliosis is a significant pathological feature of ischemic stroke and is accompanied by changes in gene expression, hypertrophied processes and proliferation. The intermediate-conductance Ca

材料
货号
品牌
产品描述

Sigma-Aldrich
TRAM-34, ≥98% (HPLC), solid