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Merck
CN

V001671

Sigma-Aldrich

钛试剂

别名:

4,5-二羟基-1,3-苯二磺酸 二钠盐, 邻苯二酚-3,5-二磺酸 二钠盐

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About This Item

经验公式(希尔记法):
C6H4Na2O8S2
CAS号:
分子量:
314.20
Beilstein:
4629740
EC 号:
MDL编号:
PubChem化学物质编号:

SMILES字符串

[Na+].[Na+].Oc1cc(cc(c1O)S([O-])(=O)=O)S([O-])(=O)=O

InChI

1S/C6H6O8S2.2Na/c7-4-1-3(15(9,10)11)2-5(6(4)8)16(12,13)14;;/h1-2,7-8H,(H,9,10,11)(H,12,13,14);;/q;2*+1/p-2

InChI key

ISWQCIVKKSOKNN-UHFFFAOYSA-L

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法律信息

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Warning

危险声明

预防措施声明

危险分类

Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3

靶器官

Respiratory system

储存分类代码

13 - Non Combustible Solids

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

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Yong Fang et al.
The Australasian journal of dermatology, 53(3), 172-180 (2012-06-28)
Free radicals and reactive oxygen species (ROS), which are generated by UV irradiation, may induce an irreversible growth arrest similar to senescence. Tiron, 4,5-dihydroxy-1,3-benzene disulfonic acid, is a widely used antioxidant to rescue ROS-evoked cell death. The aim of the
Gianluca Gortan Cappellari et al.
The Journal of nutritional biochemistry, 24(1), 371-379 (2012-11-20)
Menopause is associated with endothelial dysfunction and oxidative stress. In this condition, reduced n-3 polyunsaturated fatty acids (n-3 PUFAs) contribute to cardiovascular disease. We investigated whether treatment with n-3 PUFA reverses endothelial dysfunction and oxidative stress in experimental menopause. Thirty
Nai-Yuan Lee et al.
The Chinese journal of physiology, 53(5), 294-298 (2011-07-29)
A rise in cytosolic free Ca2+ is the immediate trigger for contraction in heart muscle. In the present study, we investigated changes of intracellular Ca2+ increased by potassium chloride (KCl) and phenylephrine (PE) under hyperglycemia in rat heart myoblast H9c2
Suresh Kumar et al.
Apoptosis : an international journal on programmed cell death, 18(5), 605-617 (2013-03-16)
This study describes the mechanism of trolox and tiron induced potentiation of cytotoxicity caused by Ery5, an analog of magnolol, in human myeloid leukemia HL-60 cells. Ery5 induced cytotoxicity in HL-60 cells by involving activation of bax and cleavage of
D D Guimarães et al.
Clinical and experimental pharmacology & physiology, 39(4), 373-378 (2012-01-31)
In pathological conditions, such as hypertension, there is impairment in the autonomic control of blood pressure resulting in changes in baroreflex sensitivity. In the present study we tested the hypothesis that acute superoxide scavenging would restore the depressed baroreflex sensitivity

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