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主要文件

Z373508

Sigma-Aldrich

Carestream® BioMax® 感光胶片

别名:

autoradiography film, xray film

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50 EA
¥5,314.03

¥5,314.03


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50 EA
¥5,314.03

About This Item

UNSPSC代码:
41105341
NACRES:
NB.22

¥5,314.03


请联系客服了解存货情况

尺寸

7 in. (18 cm) × 9.5 in. (24 cm)

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一般描述

透明基底上的双乳胶膜。为化学发光检测系统提供最清晰的背景和最高的灵敏度。也适用于放射自显影,但对高能同位素的敏感性低于X-Omat AR和BioMax MS,对中能同位素的敏感性低于BioMax MR。
Sheets of film with no interleaved paper

法律信息

BioMax is a registered trademark of Carestream Health, Inc.
Carestream is a registered trademark of Carestream Health, Inc.
Kodak is a registered trademark of Eastman Kodak Company

历史批次信息供参考:

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Matthias Höllerhage et al.
Scientific reports, 7(1), 11469-11469 (2017-09-15)
α-synuclein-induced neurotoxicity is a core pathogenic event in neurodegenerative synucleinopathies such as Parkinson's disease, dementia with Lewy bodies, or multiple system atrophy. There is currently no disease-modifying therapy available for these diseases. We screened 1,600 FDA-approved drugs for their efficacy
Shaista Naqvi et al.
Proceedings of the National Academy of Sciences of the United States of America, 106(19), 7762-7767 (2009-05-07)
Vitamin deficiency affects up to 50% of the world's population, disproportionately impacting on developing countries where populations endure monotonous, cereal-rich diets. Transgenic plants offer an effective way to increase the vitamin content of staple crops, but thus far it has
Steven C Chen et al.
PloS one, 6(5), e19780-e19780 (2011-05-24)
Although recent publications have linked the molecular events driving facioscapulohumeral muscular dystrophy (FSHD) to expression of the double homeobox transcription factor DUX4, overexpression of FRG1 has been proposed as one alternative causal agent as mice overexpressing FRG1 present with muscular
Sudhakar Raja Subramaniam et al.
Neurobiology of disease, 70, 204-213 (2014-07-13)
Parkinson's disease (PD) is characterized by the progressive degeneration of nigrostriatal dopaminergic neurons leading to motor deficits. The mechanisms underlying the preferential vulnerability of nigrostriatal dopaminergic neurons in PD remain poorly understood. Recent evidence supports a role for mitochondrial dysfunction

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