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应用
生物素标记转铁蛋白常用于加入细胞培养基，监测体外培养细胞的转铁蛋白摄取。
外形
含有柠檬酸钠的冻干粉末
制备说明
由人全铁转铁蛋白制备，通过氨基己酰基间隔基与生物素通过酰胺键偶联。
免责声明
仅供研究使用。本品在法国的科研应用受到监管，包括用于进口和出口活动（法国《公共卫生法典》Public Health Code Article L 1211-1 paragraph 2）。如《公共卫生法典》所述，购买者（即最终用户）需要从法国研究部(France Ministry of Research)获得进口授权。订购此产品即代表您已确认获得合法的进口授权。
储存分类代码
11 - Combustible Solids
WGK
WGK 3
法规信息
常规特殊物品
历史批次信息供参考:
分析证书(COA)
Lot/Batch Number
N Akhtar et al.
Molecular biology of the cell, 12(4), 847-862 (2001-04-11)
The establishment of cadherin-dependent cell-cell contacts in human epidermal keratinocytes are known to be regulated by the Rac1 small GTP-binding protein, although the mechanisms by which Rac1 participates in the assembly or disruption of cell-cell adhesion are not well understood.
Francesca Guerrieri et al.
BMC genomics, 18(1), 184-184 (2017-02-19)
The Hepatitis B Virus (HBV) HBx regulatory protein is required for HBV replication and involved in HBV-related carcinogenesis. HBx interacts with chromatin modifying enzymes and transcription factors to modulate histone post-translational modifications and to regulate viral cccDNA transcription and cellular
Marcos Francisco Núñez et al.
Molecular biology of the cell, 31(7), 667-682 (2019-12-27)
B-cells become activated by ligands with varying valency and mode of presentation to the B-cell receptor (BCR). We previously demonstrated that clustering the immunoglobulin M (IgM) isotype of BCR with an artificial soluble cross-linker stabilized an ordered phase-like domain that
Henri-François Renard et al.
Nature communications, 11(1), 1457-1457 (2020-03-21)
While several clathrin-independent endocytic processes have been described so far, their biological relevance often remains elusive, especially in pathophysiological contexts such as cancer. In this study, we find that the tumor marker CD166/ALCAM (Activated Leukocyte Cell Adhesion Molecule) is a
Dominik P Buser et al.
EBioMedicine, 46, 32-41 (2019-07-25)
Gliomas are the most frequent and aggressive malignancies of the central nervous system. Decades of molecular analyses have demonstrated that gliomas accumulate genetic alterations that culminate in enhanced activity of receptor tyrosine kinases and downstream mediators. While the genetic alterations
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