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Merck
CN

T0581

Sigma-Aldrich

转谷氨酰胺酶 来源于天竺鼠

≥1.5 units/mg protein, recombinant, expressed in E. coli

别名:

TGase

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About This Item

UNSPSC代码:
12352204
NACRES:
NA.54

重组

expressed in E. coli

质量水平

比活

≥1.5 units/mg protein

运输

dry ice

储存温度

−20°C

应用

10 mM氯化钙用于激活酶。
转谷氨酰胺酶已用于一项可改善可量化分析研究,以充分表征转谷氨酰胺酶在亨廷顿氏病和阿尔茨海默病等疾病中的作用。转谷氨酰胺酶还用于研究转谷氨酰胺酶活性的非放射性斑点印迹试验。

单位定义

一个单元,在pH6.0,37℃下,每分钟将催化Nα-Z-Gln-Gly和羟胺形成1.0 μ摩尔的异羟肟酸酯。(L-谷氨酸γ-单羟肟酸酯为参考标准。)

外形

从50 mM NaH2PO4、150 mM NaCl、pH 8冻干。包含麦芽糖糊精。

象形图

Exclamation mark

警示用语:

Warning

危险声明

危险分类

Eye Irrit. 2

储存分类代码

11 - Combustible Solids

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

常规特殊物品

从最新的版本中选择一种:

分析证书(COA)

Lot/Batch Number

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M Kelley et al.
Biochemical pharmacology, 35(2), 289-295 (1986-01-15)
The amino acid conjugation of the phenoxyherbicides 2,4-dichlorophenoxyacetate (2,4-D) and 2,4,5-trichlorophenoxyacetate (2,4,5-T) by animals was examined at the level of the enzymes catalyzing the reactions. The phenoxyherbicides were not substrates for the bile acid conjugating system but were substrates for
C M Becker et al.
Archives of biochemistry and biophysics, 223(2), 381-392 (1983-06-01)
Valproic acid (dipropylacetic acid), an antiepileptic agent known to be hepatotoxic in some patients, caused inhibition of lactate gluconeogenesis, fatty acid oxidation, and fatty acid synthesis by isolated hepatocytes. The latter process was the most sensitive to valproic acid, 50%
T S Emundianughe
Indian journal of experimental biology, 27(2), 160-162 (1989-02-01)
The metabolism of benzoic acid was examined in S. mansoni infected CBA mouse. The result showed that control animals dosed with 150 mg/kg benzoic acid resulted in urinary excretion of two metabolites, hippuric acid and benzoic acid glucuronide. Administration of
I A Qureshi et al.
Biochemistry international, 19(3), 657-666 (1989-09-01)
The development of the hepatic and renal hippurate-synthesizing system, as represented by the overall reaction of the benzoyl CoA: glycine N-acyltransferase (EC 2.3.1.13) was studied in 0, 4, 8, 13, 17, 21-day and 8-week old sparse-fur (spf) mutant mice with
K D MacDermot et al.
Developmental pharmacology and therapeutics, 3(3), 150-159 (1981-01-01)
We report investigations of benzoate and glycine metabolism and glycine acyltransferase activity in rats. These studies provide insights related to the therapy and pathophysiology of human nonketotic hyperglycinemia. Liver acyltransferase activity increased sharply postnatally from low levels at birth, but

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