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Merck
CN

SRP0444

A20 人

recombinant, expressed in baculovirus infected Sf9 cells, ≥20% (SDS-PAGE)

别名:

OTUD7C, TNFAIP3, 含有OTU结构域的蛋白7C, 肿瘤坏死因子-? 诱导蛋白3

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关于此项目

NACRES:
NA.32
UNSPSC Code:
12352202

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产品名称

A20 人, recombinant, expressed in baculovirus infected Sf9 cells, ≥20% (SDS-PAGE)

biological source

human

recombinant

expressed in baculovirus infected Sf9 cells

assay

≥20% (SDS-PAGE)

form

aqueous solution

mol wt

90.5 kDa

packaging

pkg of 50 μg

NCBI accession no.

NM_006290

UniProt accession no.

P21580

shipped in

dry ice

storage temp.

−70°C

Gene Information

human ... TNFAIP3(7128)

Application

可用于研究酶动力学、抑制剂筛选和选择性分析。

General description

人A20，也称为肿瘤坏死因子α诱导蛋白3（TNFAIP3）或含有OTU结构域的蛋白7C（OTUD7C），（GenBank登录号NM_006290）氨基酸2-791（末端），带有N-末端标记标签，MW=90.5 kDa，通过杆状病毒表达系统在Sf9昆虫细胞中表达。

存储类别

10 - Combustible liquids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

常规特殊物品
此项目有

历史批次信息供参考:

分析证书(COA)

Lot/Batch Number
8039411027

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Chong-Shan Shi et al.
Autophagy, 6(7), 986-987 (2010-08-28)
Toll-like receptor 4 (TLR4) signaling triggers autophagy, which has been linked to both adaptive and innate immunity. Engagement of TLR4 recruits to the receptor complex Beclin 1, a key component of a class III phosphatidylinositol 3-kinase complex (PI3KC3) that initiates
Jing Liu et al.
Journal of biomedical science, 20, 74-74 (2013-10-09)
Intestinal polyps may further develop into colon cancer; the pathogenesis is not clear. The p53 gene is an important anti-cancer gene in the body, which is suppressed in cancer. The ubiquitin E3 ligase A20 (A20) plays a role in regulating
Sarah G Hymowitz et al.
Nature reviews. Cancer, 10(5), 332-341 (2010-04-13)
Clinicians have suspected for hundreds of years that chronic activation of the immune system contributes to the development of cancer. However, the molecular mechanisms that mediate this precarious interplay are only now being elucidated. Recent reports have identified A20 as
Rita M Tavares et al.
Immunity, 33(2), 181-191 (2010-08-14)
A20 is a ubiquitin modifying enzyme that restricts NF-kappaB signals and protects cells against tumor necrosis factor (TNF)-induced programmed cell death. Given recent data linking A20 (TNFAIP3) with human B cell lymphomas and systemic lupus erythematosus (SLE), we have generated

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