所有图片(1)
(F13A)apelin-13, Apelin-13(F13A), Gln-Arg-Pro-Arg-Leu-Ser-His-Lys-Gly-Pro-Met-Pro-Ala TFA, L-谷氨酰胺基-L-精氨酸基-L-脯氨酰基-L-精氨酸基-L-亮氨酰-L-丝氨酰-L-组氨酸基-L-赖氨酰糖基-L-脯氨酰基-L-甲硫酰基-L-脯氨酰基-L-丙氨酸TFA, QRPRLSHKGPMPA TFA, [Ala-13] apelin-13, 丙氨酸apelin-13
C63H107N23O16S · xC2HF3O2
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检测方案
≥96% (HPLC)
形式
powder
颜色
white to beige
运输
wet ice
储存温度
−20°C
生化/生理作用
(Ala13)-Apelin-13对应于在C末端(F13A)具有Phe-Ala取代的Apelin-13,并可在培养和体内动物研究中均作为针对apelin依赖性APJ功能的拮抗剂。
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
常规特殊物品
The journal of physiological sciences : JPS, 67(3), 373-385 (2016-07-03)
The objective of this study was to explore the role of apelin in the healing of gastric lesions induced by stress. Male Wistar rats were exposed to water immersion and restraint stress (WIRS) for 6 h with or without the apelin
Endocrinology, 146(1), 231-236 (2004-10-16)
The apelin peptide is the endogenous ligand for the apelin G protein-coupled receptor. The distribution of the apelin peptides and receptor are widespread in the central nervous system and periphery, with reported roles in the hypothalamic-pituitary-adrenal axis, blood pressure regulation
Atherosclerosis, 244, 1-8 (2015-11-19)
Vascular calcification (VC) is closely related to cardiovascular events in chronic kidney disease (CKD). Apelin has emerged as a potent regulator of cardiovascular function, but its role in VC during CKD remains unknown. We determined whether apelin plays a role
American journal of physiology. Gastrointestinal and liver physiology, 310(4), G249-G261 (2015-12-19)
Apelin, an endogenous ligand for APJ receptor, has been reported to be upregulated in paraventricular nucleus (PVN) following stress. Central apelin is known to stimulate release of corticotropin-releasing factor (CRF) via APJ receptor. We tested the hypothesis that stress-induced gastrointestinal
Blood, 127(7), 908-920 (2015-12-05)
Apelin peptide and its receptor APJ are directly implicated in various physiological processes ranging from cardiovascular homeostasis to immune signaling. Here, we show that apelin is a key player in hemostasis with an ability to inhibit thrombin- and collagen-mediated platelet
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