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安全信息

SAB2101094

Sigma-Aldrich

Anti-HSP90B1 antibody produced in rabbit

affinity isolated antibody

别名:

Anti-ECGP, Anti-GP96, Anti-GRP94, Anti-Heat shock protein 90kDa β (Grp94), member 1, Anti-TRA1

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About This Item

MDL编号:
UNSPSC代码:
12352203
NACRES:
NA.41

生物来源

rabbit

质量水平

偶联物

unconjugated

抗体形式

affinity isolated antibody

抗体产品类型

primary antibodies

克隆

polyclonal

表单

buffered aqueous solution

分子量

90 kDa

种属反应性

yeast, human

浓度

0.5 mg - 1 mg/mL

技术

immunohistochemistry: suitable
western blot: suitable

UniProt登记号

运输

wet ice

储存温度

−20°C

靶向翻译后修饰

unmodified

基因信息

human ... HSP90B1(7184)

免疫原

Synthetic peptide directed towards the C terminal region of human HSP90B1

生化/生理作用

HSP90B1 belongs to the heat shock protein 90 family. It is molecular chaperone that functions in the processing and transport of secreted proteins.

序列

Synthetic peptide located within the following region: EEEPEEEPEETAEDTTEDTEQDEDEEMDVGTDEEEETAKESTAEKDEL

外形

Purified antibody supplied in 1x PBS buffer with 0.09% (w/v) sodium azide and 2% sucrose.

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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储存分类代码

10 - Combustible liquids

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

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分析证书(COA)

Lot/Batch Number

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Abhijna Ballal R et al.
Stem cell research & therapy, 15(1), 108-108 (2024-04-19)
Epilepsy affects ∼60 million people worldwide. Most antiseizure medications in the market act on voltage-gated sodium or calcium channels, indirectly modulating neurotransmitter GABA or glutamate levels or multiple targets. Earlier studies made significant efforts to directly deliver GABA into the brain
Fan Yang et al.
Journal of cellular biochemistry, 119(2), 2427-2439 (2017-09-16)
Mitochondrial dysfunction has emerged as a critical pathophysiological factor of myocardial ischemia/reperfusion (I/R) injury. A thorough understanding of mitochondrial dysfunction during I/R at the molecular level is urgently needed. One prominent microRNA, miR-410, was previously reported to be dynamically regulated

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