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Merck
CN
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安全信息

SAB1102826

Sigma-Aldrich

Anti-TM4SF19 (146-160) antibody produced in rabbit

IgG fraction of antiserum

别名:

Anti-OCTM4, Anti-Transmembrane 4 L six family member 19

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About This Item

UNSPSC代码:
12352203
NACRES:
NA.41

生物来源

rabbit

偶联物

unconjugated

抗体形式

IgG fraction of antiserum

抗体产品类型

primary antibodies

克隆

polyclonal

形式

buffered aqueous solution

分子量

antigen ~22 kDa

种属反应性

human

技术

western blot: 1:500-1:2,000

UniProt登记号

运输

dry ice

储存温度

−20°C

靶向翻译后修饰

unmodified

基因信息

一般描述

Transmembrane 4 L six family member 19 (TM4SF19) or osteoclast maturation-associated gene 4 protein (OCTM4) belongs to the transmembrane 4 L six superfamily. TM4SF19 gene is mapped to human chromosome 3q29.

免疫原

synthetic peptide corresponding to amino acids 146-160 of human TM4SF19

生化/生理作用

Transmembrane 4 L six family member 19 (TM4SF19) expression is detected in the chronic heart disease samples especially in the cells, sera and tissues. It may contribute to atherosclerosis progression and may serve as a potential marker for screening atherosclerosis.

外形

0.01M 磷酸缓冲盐溶液,pH 7.4,含 15mM 叠氮化钠。

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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WGK

WGK 2

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

常规特殊物品

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Adela Chirita Emandi et al.
Frontiers in pediatrics, 7, 270-270 (2019-07-25)
3q29 deletion syndrome is a rare disorder, causing a complex phenotype. Clinical features are variable and relatively non-specific. Our report aims to present an atypical, de novo deletion in chromosome band 3q29 in a preschool boy, first child of healthy
Yuji Kubota et al.
Nature communications, 13(1), 4063-4063 (2022-07-14)
Point-mutations of MEK1, a central component of ERK signaling, are present in cancer and RASopathies, but their precise biological effects remain obscure. Here, we report a mutant MEK1 structure that uncovers the mechanisms underlying abnormal activities of cancer- and RASopathy-associated
Li Ding et al.
Biochemical and biophysical research communications, 533(4), 1204-1211 (2020-10-17)
Atherosclerosis is a chronic vascular inflammatory disease that initially starts from an arterial intima lesion and endothelial barrier dysfunction. The purpose of this study was to investigate the role of TM4SF19, a recently identified member of the transmembrane 4L six

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