推荐产品
产品名称
Shields 和 Sang M3 昆虫培养基, With L-glutamine and potassium bicarbonate., liquid, sterile-filtered, suitable for insect cell culture
质量水平
无菌性
sterile-filtered
表单
liquid
技术
cell culture | insect: suitable
组分
glucose: 10 g/L (Dextro)
L-glutamine: 0.6 g/L
运输
ambient
储存温度
2-8°C
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应用
Shields和Sang M3昆虫培养基已用于培养果蝇细胞系、幼虫和睾丸。
为果蝇细胞系培养而开发。
生化/生理作用
昆虫组织培养基已被配制成模仿特定昆虫血淋巴的主要特性。对这些培养基配方的调查显示，它们的组成有很大的不同。当添加5-20%的热灭活胎牛血清时，发现Shields和Sang M3培养基支持黑腹果蝇细胞原代和已建立培养物的快速生长。
通常也和此产品一起购买
产品编号
说明
价格
储存分类代码
12 - Non Combustible Liquids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
动植物来源培养基
Rosalind Hale et al.
eLife, 4 (2015-02-25)
To understand how long-range patterning gradients are interpreted at the cellular level, we investigate how a gradient of expression of the Four-jointed kinase specifies planar polarised distributions of the cadherins Fat and Dachsous in the Drosophila wing. We use computational
Remigio Picone et al.
PLoS biology, 8(11), e1000542-e1000542 (2010-11-26)
Because physical form and function are intimately linked, mechanisms that maintain cell shape and size within strict limits are likely to be important for a wide variety of biological processes. However, while intrinsic controls have been found to contribute to
Shuhei Kimura
FEBS letters, 587(7), 922-929 (2013-03-05)
Spermiogenesis is a dynamic process leading to alterations in cell morphology. In spermiogenesis, the roles of the histone chaperones are largely unknown. Here, I report the unexpected roles of two Nap family proteins, CG5017/Hanabi and nucleosome assembly protein 1 (Nap1)
Arthur Luhur et al.
G3 (Bethesda, Md.), 10(12), 4541-4551 (2020-10-09)
Successful Drosophila cell culture relies on media containing xenogenic components such as fetal bovine serum to support continuous cell proliferation. Here, we report a serum-free culture condition that supports the growth and proliferation of Drosophila S2R+ and Kc167 cell lines.
Giri Raj Dahal et al.
Development (Cambridge, England), 144(15), 2771-2783 (2017-07-08)
Loss of embryonic ion channel function leads to morphological defects, but the underlying reason for these defects remains elusive. Here, we show that inwardly rectifying potassium (Irk) channels regulate release of the
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