推荐产品
一般描述
活化的链霉亲和素（SA）和辣根过氧化物酶（HRP）与聚合物主链共价缀合。每个聚合物链上的多个活性生物分子可增加生物素结合能力并放大过氧化物酶信号。
特异性
生物素化的生物分子。
应用
检测生物素化的蛋白质和核酸以及其他生物分子。
适用于蛋白质印迹和ELISA，可用于检测免疫组织化学和免疫细胞化学中的表面抗原。
适用于蛋白质印迹和ELISA，可用于检测免疫组织化学和免疫细胞化学中的表面抗原。
外形
以1.0mg/ml溶液提供，溶于0.01M磷酸钠（pH 7.4），0.15M氯化钠，50％甘油，稳定剂和防腐剂中。
储存分类代码
10 - Combustible liquids
WGK
WGK 1
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
常规特殊物品
从最新的版本中选择一种：
分析证书(COA)
Lot/Batch Number
Natalia Gruba et al.
International journal of molecular sciences, 20(7) (2019-03-31)
Kallikrein 13 (KLK13) was first identified as an enzyme that is downregulated in a subset of breast tumors. This serine protease has since been implicated in a number of pathological processes including ovarian, lung and gastric cancers. Here we report
Aya Sugyo et al.
Cancers, 12(12) (2020-12-10)
In treatment-refractory cancers, tumor tissues damaged by therapy initiate the repair response; therefore, tumor tissues must be exposed to an additional burden before successful repair. We hypothesized that an agent recognizing a molecule that responds to anticancer treatment-induced tissue injury
Stefan Kaluz et al.
Biochemical and biophysical research communications, 370(4), 613-618 (2008-04-12)
The hypoxia-inducible factor (HIF) activates transcription via binding to the highly variable hypoxia-responsive elements (HREs). All hypoxia-inducible constructs described to date utilize multimers of naturally occurring HREs. Here, we describe the rational design of minimal hypoxia-inducible enhancers, conceptually equivalent to
Jaclyn C Law et al.
Journal of immunology (Baltimore, Md. : 1950), 206(1), 37-50 (2020-11-20)
There is a pressing need for an in-depth understanding of immunity to SARS-CoV-2. In this study, we investigated human T cell recall responses to fully glycosylated spike trimer, recombinant N protein, as well as to S, N, M, and E
Antonios Psarras et al.
Nature communications, 11(1), 6149-6149 (2020-12-03)
Autoimmune connective tissue diseases arise in a stepwise fashion from asymptomatic preclinical autoimmunity. Type I interferons have a crucial role in the progression to established autoimmune diseases. The cellular source and regulation in disease initiation of these cytokines is not
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