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Merck
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安全信息

RAB1162

Sigma-Aldrich

大鼠肾素1 ELISA试剂盒

for cell culture supernatants, plasma, and serum samples

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UNSPSC代码:
41116158
NACRES:
NA.32

种属反应性

rat

技术

ELISA: suitable

assay range

inter-assay cv: <12%
intra-assay cv: <10%
sensitivity: 68 pg/ml

运输

wet ice

储存温度

−20°C

一般描述

This ELISA antibody pair detects Rat Renin (REN)

应用

For research use only. Not for use in diagnostic procedures.
Please refer to the attached Protocolfor details.

其他说明

A sample Certificate of Analysis is available for this product. Please type the word sample in the text box provided for lot number.

象形图

Corrosion

警示用语:

Warning

危险声明

预防措施声明

危险分类

Met. Corr. 1

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

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Marite Punapart et al.
Genes, 12(11) (2021-11-28)
Wolfram syndrome (WS) 1 is a rare monogenic neurodegenerative disorder caused by mutations in the gene encoding WFS1. Knowledge of the pathophysiology of WS is incomplete and to date, there is no treatment available. Here, we describe early deviations in
J Oh et al.
Nature communications, 11(1), 4798-4798 (2020-09-25)
Myeloid cells are known mediators of hypertension, but their role in initiating renin-induced hypertension has not been studied. Vitamin D deficiency causes pro-inflammatory macrophage infiltration in metabolic tissues and is linked to renin-mediated hypertension. We tested the hypothesis that impaired
Junda Hu et al.
Acta physiologica (Oxford, England), 232(1), e13612-e13612 (2020-12-31)
The use of calcineurin inhibitors such as cyclosporine A (CsA) for immunosuppression after solid organ transplantation is commonly limited by renal side effects. CsA-induced deterioration of glomerular filtration rate and sodium retention may be related to juxtaglomerular dysregulation as a
Dong Cheng et al.
Molecular medicine reports, 22(5), 4151-4162 (2020-10-02)
Clinical application of doxorubicin (DOX) is hampered by its potential cardiotoxicity, however angiotensin receptor blockers could attenuate DOX‑induced cardiomyopathy. The present study tested the hypothesis that simultaneous administration of valsartan (Val) with DOX could prevent DOX‑induced myocardial injury by modulating myocardial NAD(P)H

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