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种属反应性
human
包装
kit of 96 wells (12 strips x 8 wells)
技术
ELISA: suitable
capture ELISA: suitable
输入
sample type serum
sample type cell culture supernatant(s)
sample type urine
sample type plasma
assay range
inter-assay cv: <12%
intra-assay cv: <10%
sensitivity: 20 pg/mL
standard curve range: 24.58-6000 pg/mL
检测方法
colorimetric
运输
wet ice
储存温度
−20°C
基因信息
human ... CCL7(6354)
一般描述
The Human MCP-3 (Monocyte Chemoattractant Protein-3) ELISA (Enzyme-Linked Immunosorbent Assay) kit is an in vitro enzyme- linked immunosorbent assay for the quantitative measurement of human MCP-3 in serum, plasma, cell culture supernatants and urine.
免疫原
Recombinant Human MCP3
应用
请参考Protocol了解详情。
生化/生理作用
Monocyte-chemotactic protein 3 (MCP3), also known as chemokine (C-C motif) ligand 7 (CCL7), has an ability to attract and activate a large panel of leukocytes including monocytes, eosinophils, basophils, dendritic cells, natural killer cells and activated T lymphocytes. This property of the protein makes it favorable for antitumor therapy. Mutation in the gene is associated with the development of Crohn′s Disease (CD). Elevated expression of the gene contributes to the development of gastric cancer (GC). Thus, it acts as a potent diagnostic marker for GC.
其他说明
A sample Certificate of Analysis is available for this product.
Please type the word sample in the text box provided for lot number.
Please type the word sample in the text box provided for lot number.
试剂盒组分也可单独购买
产品编号
说明
化学品安全说明书
警示用语：
Warning
危险声明
预防措施声明
危险分类
Met. Corr. 1
储存分类代码
8A - Combustible corrosive hazardous materials
法规信息
新产品
MCP?3 (CCL7) delivered by parvovirus MVMp reduces tumorigenicity of mouse melanoma cells through activation of T lymphocytes and NK cells
Wetzel K, et al.
International Journal of Cancer. Journal International Du Cancer, 120(6), 1364-1371 (2007)
CCL7 and CCL21 overexpression in gastric cancer is associated with lymph node metastasis and poor prognosis
Hwang TL, et al.
World Journal of Gastroenterology, 18(11), 1249-1249 (2012)
The Genomics of Autoimmune Disease in the Era of Genome-Wide Association Studies and Beyond
Lessard CJ, et al.
Autoimmunity Reviews, 11(4), 267-275 (2012)
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