推荐产品
类型
Type I
质量水平
方案
≥98% (TLC)
表单
lyophilized powder
脂质类型
phosphoglycerides
储存温度
−20°C
SMILES字符串
[P](=O)([O-])(OCC[N+H3])OCC(OC(=O)C)COC(=O)C
InChI
1S/C9H18NO8P/c1-7(11)15-5-9(18-8(2)12)6-17-19(13,14)16-4-3-10/h9H,3-6,10H2,1-2H3,(H,13,14)
InChI key
CFWRDBDJAOHXSH-UHFFFAOYSA-N
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生化/生理作用
脑中的主要结构磷脂，构成总磷脂的 20-25%；主要固定于灰质上。
包装
在氩气下包装。
注意
制备时纯度为 98-99%。该溶液暴露在室温下每天可分解 0.3-0.5%。
历史批次信息供参考:
分析证书(COA)
Lot/Batch Number
Yanfeng Zhang et al.
Biochemical and biophysical research communications, 404(1), 407-412 (2010-12-07)
The botulinum neurotoxins (BoNTs) produced by different strains of the bacterium Clostridium botulinum are responsible for the disease botulism and include a group of immunologically distinct serotypes (A, B, E, and F) that are considered to be the most lethal
Ryozo Azuma et al.
International journal of systematic and evolutionary microbiology, 59(Pt 6), 1469-1473 (2009-06-09)
A Gram-positive, short diphtheroid-shaped organism was isolated from a sow's placenta of an abortion. This novel isolate, strain Murakami(T), was examined physiologically, chemotaxonomically and phylogenetically. Cells had an irregular V-shaped or palisade arrangement. Colonies appeared translucent on TMVL agar. Cells
Daniel E Otzen et al.
Biochimica et biophysica acta, 1818(4), 1019-1027 (2011-09-17)
Lactadherin binds to phosphatidylserine (PS) in a stereospecific and calcium independent manner that is promoted by vesicle curvature. Because membrane binding of lactadherin is supported by a PS content of as little as 0.5%, lactadherin is a useful marker for
Kelly Varga et al.
Journal of neurochemistry, 152(1), 48-60 (2019-10-07)
Phosphatidylserine (PS), a negatively charged phospholipid present predominantly at the inner leaflet of the plasma membrane, has been widely implicated in many cellular processes including membrane trafficking. Along this line, PS has been demonstrated to be important for endocytosis, however
Shane P Herbert et al.
The Journal of biological chemistry, 284(9), 5784-5796 (2009-01-03)
Release of endothelial cells from contact-inhibition and cell cycle re-entry is required for the induction of new blood vessel formation by angiogenesis. Using a combination of chemical inhibition, loss of function, and gain of function approaches, we demonstrate that endothelial
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