所有图片(4)
别名:
PC, (7R)-4-羟基-N,N,N-三甲基-10-氧代-7-[(1-氧代十六烷基)氧基]- 3,5,9-三氧杂-4-磷杂二十五烷-1-铵4-氧化物， 内盐, 1,2-二十六酰- 锡 -甘油基-3-磷酸胆碱, 1,2-二棕榈酰-sn-甘油-3-磷酰胆碱, 1,2-二棕榈酰- L -卵磷脂, 1,2-双十六酰基-sn-甘油-3-磷酰胆碱, L-α-磷脂酰胆碱，二棕榈酰, L-β,γ-二棕榈酰-α-卵磷脂, PC(16:0/16:0), 二棕榈酰磷脂酰胆碱
经验公式（希尔记法）:
C40H80NO8P
CAS号:
分子量:
734.04
EC 号:
MDL编号:
UNSPSC代码:
12352211
PubChem化学物质编号:
NACRES:
NA.25
推荐产品
生物来源
synthetic
质量水平
检测方案
≥99% (TLC)
形式
powder
官能团
phospholipid
脂质类型
phosphoglycerides
运输
ambient
储存温度
−20°C
SMILES字符串
[O-]P(OCC[N+](C)(C)C)(OC[C@]([H])(OC(CCCCCCCCCCCCCCC)=O)COC(CCCCCCCCCCCCCCC)=O)=O
InChI
1S/C40H80NO8P/c1-6-8-10-12-14-16-18-20-22-24-26-28-30-32-39(42)46-36-38(37-48-50(44,45)47-35-34-41(3,4)5)49-40(43)33-31-29-27-25-23-21-19-17-15-13-11-9-7-2/h38H,6-37H2,1-5H3/t38-/m1/s1
InChI key
KILNVBDSWZSGLL-KXQOOQHDSA-N
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应用
- Effects induced by (6)-p-cymene ruthenium(II) complexes on Langmuir monolayers mimicking cancer and healthy cell membranes do not correlate with their toxicity.: This study investigates the interaction of ruthenium(II) complexes with Langmuir monolayers, including 1,2-Dipalmitoyl-sn-glycero-3-phosphocholine, highlighting differences in behavior between cancerous and healthy cell membrane models (Wrobel et al., 2024).
- Cholesterol Changes Interfacial Water Alignment in Model Cell Membranes.: This study examines how cholesterol affects water alignment at the interface of model cell membranes that include 1,2-Dipalmitoyl-sn-glycero-3-phosphocholine, offering insights into membrane biophysics (Orlikowska-Rzeznik et al., 2024).
- Impact of Lipid Composition on Vesicle Protein Adsorption: A BSA Case Study.: The impact of different lipid compositions, including 1,2-Dipalmitoyl-sn-glycero-3-phosphocholine, on the adsorption of bovine serum albumin to vesicles is studied to understand protein-lipid interactions (Amarandi et al., 2024).
- Spatial Arrangement of the Drug Ibuprofen in a Model Membrane in the Presence of Lipid Rafts.: This paper investigates the arrangement of ibuprofen within model membranes containing lipid rafts and 1,2-Dipalmitoyl-sn-glycero-3-phosphocholine, shedding light on drug-membrane interactions (Kashnik et al., 2024).
联系
通过结晶纯化。
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Johannes Thoma et al.
Chemical communications (Cambridge, England), 48(70), 8811-8813 (2012-07-28)
We present a model system to demonstrate that the positioning of biomolecules (membrane proteins) in a nonnative, complex thin film environment can be regulated by the phase behavior of film components. Partial separation between an amphiphilic polymer and a lipid
Edyta Paszko et al.
European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences, 48(1-2), 202-210 (2012-11-20)
Photodynamic therapy (PDT) is based on the delivery of photocytotoxic agents to a target tissue, followed by irradiation. In order to increase the efficiency of PDT in oesophageal cancer therapy, polyethylene glycol (PEG)-grafted, transferrin (Tf)-conjugated liposome formulations of 5,10,15,20-tetra(m-hydroxyphenyl)chlorin (Foscan)
Fernando R Moya et al.
Pediatrics, 115(4), 1018-1029 (2005-04-05)
Evidence suggests that synthetic surfactants consisting solely of phospholipids can be improved through the addition of peptides, such as sinapultide, that mimic the action of human surfactant protein-B (SP-B). A synthetic surfactant containing a mimic of SP-B may also reduce
John M Franck et al.
Journal of the American Chemical Society, 135(11), 4175-4178 (2013-01-26)
The translational hydration dynamics within 0.5-1.5 nm of the surface of a DPPC liposome, a model biomacromolecular surface, is analyzed by the recently developed Overhauser dynamic nuclear polarization (ODNP) technique. We find that dramatic changes to the bulk solvent cause
Xu Hannah Zhang et al.
Journal of cell science, 120(Pt 23), 4134-4143 (2007-11-23)
The G1 phase of the cell cycle is marked by the rapid turnover of phospholipids. This turnover is regulated by CTP:phosphocholine-cytidylyltransferase (CCT) and group VIA Ca(2+)-independent-phospholipase A(2) (iPLA(2)). We previously reported that inhibition of iPLA(2) arrests cells in G1 phase
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