推荐产品
产品名称
4-甲基伞形酮基β-D-葡萄糖苷, β-glucosidase substrate
质量水平
方案
≥99% (HPLC)
表单
powder
溶解性
DMF: 50 mg/mL, clear, colorless to faintly yellow
荧光
λex 316 nm; λem 372 nm (pH 9.1)
λex 360 nm; λem 449 nm (Reaction product)
储存温度
−20°C
SMILES字符串
CC1=CC(=O)Oc2cc(O[C@@H]3O[C@H](CO)[C@@H](O)[C@H](O)[C@H]3O)ccc12
InChI
1S/C16H18O8/c1-7-4-12(18)23-10-5-8(2-3-9(7)10)22-16-15(21)14(20)13(19)11(6-17)24-16/h2-5,11,13-17,19-21H,6H2,1H3/t11-,13-,14+,15-,16-/m1/s1
InChI key
YUDPTGPSBJVHCN-YMILTQATSA-N
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应用
4-甲基伞形酮基β-D-葡萄糖苷可用来作为以下反应的底物:
- 原发性海马神经元富含溶菌酶部分葡糖神经酰胺酶β 酶活性分析
- 酵母发酵过程中β-葡萄糖苷酶分析
- 巨噬细胞细胞系(RAW)中的葡萄糖脑苷脂酶1(GBA1)-相关葡萄糖苷酶活性分析
生化/生理作用
4-甲基伞形酮基β-D-葡萄糖苷是一种合成糖苷酶底物。它可用作来自肠球菌的葡萄糖苷酶底物。
储存分类代码
11 - Combustible Solids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Eyeshields, Gloves, type N95 (US)
法规信息
监管及禁止进口产品
从最新的版本中选择一种:
分析证书(COA)
Assessing microbial safety of drinking water improving approaches and methods: Improving approaches and methods, 253-253 (2003)
Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation, 21(6), 1059-1067 (2015-02-25)
This retrospective report compared the 4-year outcomes of allogeneic stem cell transplantation (allo-SCT) in 651 adult patients with acute myeloid leukemia receiving a reduced-intensity (RIC) or nonmyeloablative conditioning (NMA) regimen according to the type of unrelated donors. These were either
Determinants on an efficient cellulase recycling process for the production of bioethanol from recycled paper sludge under high solid loadings
Biotechnology for Biofuels, 11(1), 111-111 (2018)
Nature communications, 11(1), 3327-3327 (2020-07-06)
Gaucher disease is a lysosomal storage disorder caused by insufficient glucocerebrosidase activity. Its hallmark manifestations are attributed to infiltration and inflammation by macrophages. Current therapies for Gaucher disease include life-long intravenous administration of recombinant glucocerebrosidase and orally-available glucosylceramide synthase inhibitors.
Mitochondrial dysfunction and mitophagy defect triggered by heterozygous GBA mutations
Autophagy, 15(1), 113-130 (2019)
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