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生化/生理作用
荆豆凝集素具有抗H血型特异性。凝集素有两种类型： UEA I 对 L-岩藻糖具有亲和力，而 UEA II 对 N,N′-二乙酰壳二糖具有亲和力。最初认为UEA I摩尔量为170,000道尔顿。 后来的报道表明，UEA I可以在中性和碱性pH下形成聚集体，正确的摩尔量是68,000道尔顿。
包装
包装规格基于蛋白含量。
联系
由亲和纯化的凝集素制备缀合物。
外形
含有磷酸钾缓冲盐和NaCl
分析说明
凝集活性以μg/ml表示，由磷酸盐缓冲盐水（pH 6.8）连续稀释1 mg/ml溶液而测得。该活性是指25℃孵育1小时后使2％人O型血红细胞悬浮液凝集的最低浓度。
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Eyeshields, Gloves, type N95 (US)
法规信息
动植物源性产品
Karla Lehle et al.
Artificial organs, 40(6), 577-585 (2015-10-30)
Multipotent progenitor cells were mobilized during pediatric extracorporeal membrane oxygenation (ECMO). We hypothesize that these cells also adhered onto polymethylpentene (PMP) fibers within the membrane oxygenator (MO) during adult ECMO support. Mononuclear cells were removed from the surface of explanted
Naoki Akanuma et al.
Pancreas, 46(9), 1202-1207 (2017-09-14)
We aimed to evaluate the contribution of acinar-to-ductal metaplasia (ADM) to the accumulation of cells with a ductal phenotype in cultured human exocrine pancreatic tissues and reveal the underlying mechanism. We sorted and cultured viable cell populations in human exocrine
Zoe E Clayton et al.
International journal of cardiology, 234, 81-89 (2017-02-18)
Endothelial cells derived from human induced pluripotent stem cells (iPSC-ECs) promote angiogenesis, and more recently induced endothelial cells (iECs) have been generated via fibroblast trans-differentiation. These cell types have potential as treatments for peripheral arterial disease (PAD). However, it is
Alan Pestronk et al.
Muscle & nerve, 42(1), 53-61 (2010-06-15)
The causes of perifascicular myofiber atrophy and capillary pathology in dermatomyositis are incompletely understood. We studied 11 dermatomyositis muscles by histochemistry, immunohistochemistry, and ultrastructure. We found that endomysial capillaries within regions of perifascicular atrophy are not entirely lost, but they
Gang Chen et al.
The Journal of clinical investigation, 119(10), 2914-2924 (2009-09-18)
Various acute and chronic inflammatory stimuli increase the number and activity of pulmonary mucus-producing goblet cells, and goblet cell hyperplasia and excess mucus production are central to the pathogenesis of chronic pulmonary diseases. However, little is known about the transcriptional
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