产品名称
Knudson C 改良兰花培养基, powder, suitable for plant cell culture
form
powder
technique(s)
cell culture | plant: suitable
application(s)
agriculture
storage temp.
2-8°C
Quality Level
Application
Knudson C 改良兰花培养基是兰科植物种子萌发的营养液。这种培养基通常添加 100-150g 香蕉，或 100-200 mL 椰子汁，每升 2g 蔬菜炭。
Preparation Note
signalword
Danger
hcodes
Hazard Classifications
Eye Dam. 1
存储类别
11 - Combustible Solids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
动植物来源培养基
此项目有
Philip J Kauth et al.
Annals of botany, 102(5), 783-793 (2008-09-02)
Ecotypic differentiation has been explored in numerous plant species, but has been largely ignored in the Orchidaceae. Applying a specific germination protocol for widespread seed sources may be unreliable due to inherent physiological or genetic differences in localized populations. It
Guek Eng Sim et al.
Plant cell reports, 26(4), 383-393 (2006-10-07)
We have successfully developed a method to induce early in vitro flowering of the self-pollinated seedlings of a tropical orchid hybrid, Dendrobium Madame Thong-In. Transition of vegetative shoot apical meristem to inflorescence meristem was observed when young protocorms were cultured
E Ouni et al.
Human reproduction (Oxford, England), 35(6), 1391-1410 (2020-06-17)
How do elastic matrisome components change during the lifetime of the human ovary? The deposition and remodeling of mechanical matrisome components (collagen, elastin, elastin microfibril interface-located protein 1 (EMILIN-1), fibrillin-1 and glycosaminoglycans (GAGs)) that play key roles in signaling pathways
Kate Lykke Lambertsen et al.
PloS one, 7(10), e47744-e47744 (2012-10-19)
The calmodulin/calcium-activated K(+) channel KCa3.1 is expressed in red and white blood cells, epithelia and endothelia, and possibly central and peripheral neurons. However, our knowledge about its contribution to neurological functions and behavior is incomplete. Here, we investigated whether genetic
Giovanni Di Liberto et al.
Cell, 175(2), 458-471 (2018-09-04)
Inflammatory disorders of the CNS are frequently accompanied by synaptic loss, which is thought to involve phagocytic microglia and complement components. However, the mechanisms accounting for aberrant synaptic connectivity in the context of CD8+ T cell-driven neuronal damage are poorly understood.
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