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Merck
CN
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文件

安全信息

GHS232

Sigma-Aldrich

苏木精,Gill 2 号

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MDL编号:
UNSPSC代码:
12352200
NACRES:
NA.47

形式

solution

质量水平

保质期

Expiry date on the label.

IVD

for in vitro diagnostic use

浓度

4 g/L

pH值(酸碱度)

2.5-3.5

应用

hematology
histology

储存温度

room temp

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应用

Gill配方2可用于细胞学和/或组织学,基于染色时间长度用作进行性或退行性染色。

其他说明

4 g/L 认证型苏木精

象形图

Corrosion

警示用语:

Danger

危险声明

预防措施声明

危险分类

Eye Dam. 1

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

法规信息

监管及禁止进口产品

分析证书(COA)

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Joan E Hooper et al.
Frontiers in physiology, 11, 1099-1099 (2020-10-06)
Alternative splicing acts as a fundamental mechanism to increase the number of functional transcripts that can be derived from the genome - and its appropriate regulation is required to direct normal development, differentiation, and physiology, in many species. Recent studies
Xuemei Liu et al.
Scientific reports, 7(1), 2762-2762 (2017-06-07)
Chronic treatment with second-generation antipsychotic drugs (SGAs) has been associated with an increased risk of metabolic syndrome. To evaluate the longitudinal changes in glucose-lipid homeostasis after SGA use, we studied the time-dependent effects of olanzapine (OLZ) (3 mg/kg, b.i.d.) or clozapine
Helen F Irving-Rodgers et al.
Cell and tissue research, 339(3), 613-624 (2009-12-25)
Despite the mouse being an important laboratory species, little is known about changes in its extracellular matrix (ECM) during follicle and corpora lutea formation and regression. Follicle development was induced in mice (29 days of age/experimental day 0) by injections
Beth G Ashinsky et al.
Acta biomaterialia, 111, 232-241 (2020-05-25)
Tissue-engineered replacement discs are an area of intense investigation for the treatment of end-stage intervertebral disc (IVD) degeneration. These living implants can integrate into the IVD space and recapitulate native motion segment function. We recently developed a multiphasic tissue-engineered disc-like
Michelle Naughton et al.
Journal of neuroinflammation, 17(1), 349-349 (2020-11-24)
Multiple sclerosis (MS) is an immune-mediated disease that damages myelin in the central nervous system (CNS). We investigated the profile of CCN3, a known regulator of immune function and a potential mediator of myelin regeneration, in multiple sclerosis in the

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