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Merck
CN

GENA935

ECL Rat IgG, HRP-linked whole antibody (from goat)

Cytiva NA935

别名:

Rat IgG antibody, HRP-conjugated secondary antibody, anti-rat IgG HRP, goat anti-rat, peroxidase-linked anti-rat

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关于此项目

NACRES:
NA.46
UNSPSC Code:
12352203
Clone:
-
Species reactivity:
-
Application:
Citations:
5
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antibody form

Ig fraction of antiserum

antibody product type

primary antibodies

feature

wetted part: no

packaging

pkg of 1 mL

manufacturer/tradename

Cytiva NA935

color

Brown (light)

shipped in

wet ice

storage temp.

2-8°C

General description

The antibody is prepared by hyper-immunizing goats with purified immunoglobulin fractions from normal rat serum to produce high affinity antibodies. The pooled antiserum is used to produce an immunoglobulin preparation which is then affinity adsorbed to remove cross-reacting antibodies towards mouse, human and rabbit immunoglobulins. These activities are thoroughly depleted to ensure species-specificity. Finally, to select for specific binding to rat IgG, the antibodies are purified using an affinity column of rat IgG. After washing to remove non-specific serum components and low affinity antibodies, the species-specific antibodies are eluted using carefully selected, mild conditions which minimize aggregation and preserve immunological activity, yet which will elute high affinity antibodies.

Features and Benefits

  • Highly species-specific.
  • Optimized for use with our range of ECL Detection Reagents.
  • Recommended dilutions to minimize background.

Preparation Note

Please be aware this product may be shipped 90 days before the expiration date. For more information on the batch specific expiration date, please contact technical service.

Analysis Note

To view the Certificate of Analysis for this product, please visit www.cytiva.com.

Legal Information

ECL is a trademark of Cytiva

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存储类别

12 - Non Combustible Liquids

法规信息

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历史批次信息供参考:

分析证书(COA)

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Elisabet Barbero-Camps et al.
Autophagy, 14(7), 1129-1154 (2018-06-05)
Macroautophagy/autophagy failure with the accumulation of autophagosomes is an early neuropathological feature of Alzheimer disease (AD) that directly affects amyloid beta (Aβ) metabolism. Although loss of presenilin 1 function has been reported to impair lysosomal function and prevent autophagy flux
Magnus Stårsta et al.
PLoS genetics, 16(2), e1008607-e1008607 (2020-02-14)
RHS elements are components of conserved toxin-delivery systems, wide-spread within the bacterial kingdom and some of the most positively selected genes known. However, very little is known about how Rhs toxins affect bacterial biology. Salmonella Typhimurium contains a full-length rhs
Kristin A Knouse et al.
Cell, 175(1), 200-211 (2018-08-28)
Much of our understanding of chromosome segregation is based on cell culture systems. Here, we examine the importance of the tissue environment for chromosome segregation by comparing chromosome segregation fidelity across several primary cell types in native and nonnative contexts.
Waqas Azeem et al.
Biomedicines, 9(8) (2021-08-28)
Modulation of β-catenin signaling has attractive therapeutic potential in cancer immunotherapy. Several studies have found that β-catenin can mediate immune evasion in cancer and promote anti-inflammatory features of antigen-presenting dendritic cells. Many small molecular compounds that inhibit Wnt/β-catenin signaling are
Vicente Roca-Agujetas et al.
Molecular neurodegeneration, 16(1), 15-15 (2021-03-10)
Emerging evidence indicates that impaired mitophagy-mediated clearance of defective mitochondria is a critical event in Alzheimer's disease (AD) pathogenesis. Amyloid-beta (Aβ) metabolism and the microtubule-associated protein tau have been reported to regulate key components of the mitophagy machinery. However, the

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