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Merck
CN

EMU147821

MISSION® esiRNA

targeting mouse Mgrn1

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NACRES:
NA.51
UNSPSC Code:
41105324

主要文件

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产品名称

MISSION® esiRNA, targeting mouse Mgrn1

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

TGTCTGCACGCCTCATGTGGCCACTGGGATTTGAGGGTCCACAGTGAACCTTGATCAAATTGCACAGTGGGTTGTTCCTCCACACACCCTCCTTTTTTTACGGTCAATATATTTGTAAATGGTACAAGATGAAGGACAACAACTGTCTCCATGGGCCACGTGCCCTAGAAGGACTCTCCCCAACTGTCAAAGCTAATGCTTTGAGCACTTAGCAAACCAGCATAAGAGAATCTACCCATGGAATGTTCTGGAAGGTGGGCAGTCCAGGTTACAGTTGATGAAGGAAACAGTGCCTGTCTGCCTCTCATGTGTGACCCCAAGGCAGCCAGGCTGTCACTGCTGCTGTCACAGGCTGACACCTTACAGATGTCCTGACCATGGGTGACCCTCCTCGGAGCGTCCAGATCCCCTCCTGCTCTGCTCTATTACCTACTAGTGGTCCCTGGCAGCTGTGGGCAAGGCCTGGGTAGTCAGACTGTTAACACCACAGTCCTTAGCAGTGGTCAATGTCCAGTT

Ensembl | mouse accession no.

ENSMUSG00000022517

NCBI accession no.

NM_029657

shipped in

ambient

storage temp.

−20°C

Quality Level

100

Gene Information

mouse ... MGRN1(17237), Mgrn1(17237)

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

存储类别

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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分析证书(COA)

Lot/Batch Number
BWU
JFS

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Deepak Chhangani et al.
Biochimica et biophysica acta, 1842(9), 1472-1484 (2014-04-29)
Polyglutamine diseases are a family of inherited neurodegenerative diseases caused by the expansion of CAG repeats within the coding region of target genes. Still the mechanism(s) by which polyglutamine proteins are ubiquitinated and degraded remains obscure. Here, for the first
Ye Chun Ruan et al.
Journal of cell science, 127(Pt 20), 4396-4408 (2014-08-12)
Mutations in CFTR lead to dysfunction of tubular organs, which is currently attributed to impairment of its conductive properties. We now show that CFTR regulates tight junction assembly and epithelial cell differentiation through modulation of the ZO-1-ZONAB pathway. CFTR colocalizes

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