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Merck
CN

EMU070451

MISSION® esiRNA

targeting mouse Mmab

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NACRES:
NA.51
UNSPSC Code:
41105324

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产品名称

MISSION® esiRNA, targeting mouse Mmab

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

AGAGACGTGTGGTGCCTCTTGTCCAGATGGGAGAGACGGATGCAAACGTGGCGAAGTTCTTAAACAGGCTCAGTGATTACCTCTTCACTGTAGCCAGATATGCAGCCATGAAGGAGGGCAGCCAAGAGAAAATATACAAGAAACATGATGTGTGAAGCATGAATTTGCACAGTGGGCGAGAGGTCAACTCCAGACAGTCGTGCTGAGAGTGGAAGTGGGGGTTGGCTACAGACTCAGCATCACCTTGTGCTCACCCCCCCGCCCTCCAGGGACTTTCGAGACTCCTGGCTTCTCCAGGTTGTGTCGAACATGTCAGCTGACTTCCAAATTCTGGGAAGGCAAGGTAAACTGGAGGCCACGGAGGGGATGGGCTCCTCGGAATTGAGAATGAACATGGGGAAGGTTTGAACTGTGCACTTGGGCTTTCT

Ensembl | mouse accession no.

ENSMUSG00000029575

NCBI accession no.

NM_029956

shipped in

ambient

storage temp.

−20°C

Quality Level

100

Gene Information

mouse ... MMAB(77697), Mmab(77697)

相关类别

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

存储类别

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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Amit Kumar et al.
PloS one, 9(6), e100228-e100228 (2014-06-28)
The Kaposi's sarcoma-associated herpesvirus infects the human population and maintains latency stage of viral life cycle in a variety of cell types including cells of epithelial, mesenchymal and endothelial origin. The establishment of latent infection by KSHV requires the expression
Maude Gabriel et al.
BMC cancer, 15, 227-227 (2015-04-18)
Modification of splicing by chemotherapeutic drugs has usually been evaluated on a limited number of pre-mRNAs selected for their recognized or potential importance in cell proliferation or apoptosis. However, the pathways linking splicing alterations to the efficiency of cancer therapy

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