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Merck
CN

EHU079951

MISSION® esiRNA

targeting human PLAGL1

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NACRES:
NA.51
UNSPSC Code:
41105324

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产品名称

MISSION® esiRNA, targeting human PLAGL1

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

TGGAAAACGAGACTGGGACTATGGCTTATTCAGTGATGACTGGCTTGAGATGATAAGAGAATTCTCGAACTGCATGTATTGTGCCAATCTGTCCTGAGTGTTCATGCTTTGTACCAAATTTAATGAACGCGTGTTCTGTAATCAAACTGCAAATATTGTCATAACCAACATCCAAAATGACGGCTGCTATATATAAGTGTTTGTCATATGGAATTTAATCGTAAGCCATGATCATAATGTTAACTAAATAACTTTATGTGGCACTGCCTAGTAAGGGAACTATGGAAAGGTTTGGATTTCTCCAAATCTGGGAGAATTTTCAAAATAAGAAAATAACCTTTATATGATATACTATGACTAGGCTGTGTATTTCTTTTCAGGGATTTTTCTACCTTCAGGGTTGGA

Ensembl | human accession no.

ENSG00000118495

NCBI accession no.

NM_002656

shipped in

ambient

storage temp.

−20°C

Quality Level

100

Gene Information

human ... PLAGL1(5325), PLAGL1(5325)

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

存储类别

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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Rebekah R Starks et al.
International journal of molecular sciences, 21(21) (2020-11-12)
During pregnancy, the placenta is important for transporting nutrients and waste between the maternal and fetal blood supply, secreting hormones, and serving as a protective barrier. To better understand placental development, we must understand how placental gene expression is regulated.
Yeyou Liang et al.
Metabolism: clinical and experimental, 64(12), 1682-1693 (2015-10-13)
Evidence shows that both macrophage migration inhibitory factor (MIF) and GLUT4 glucose transporter are involved in diabetic cardiomyopathy (DCM), but it remains largely unknown whether and how MIF regulates GLUT4 expression in cardiomyocytes. The present study aims to investigate the

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