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Merck
CN

C8383

Sigma-Aldrich

氯醛 水合物

别名:

三氯乙醛 水合物

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About This Item

线性分子式:
Cl3CCH(OH)2
CAS号:
分子量:
165.40
Beilstein:
1698497
EC 号:
MDL编号:
UNSPSC代码:
51111800
PubChem化学物质编号:
NACRES:
NA.77

方案

≥98% (titration)

质量水平

表单

powder

药品控制

USDEA Schedule IV

mp

57 °C (lit.)

SMILES字符串

OC(O)C(Cl)(Cl)Cl

InChI

1S/C2H3Cl3O2/c3-2(4,5)1(6)7/h1,6-7H

InChI key

RNFNDJAIBTYOQL-UHFFFAOYSA-N

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生化/生理作用

氯水合物是镇静剂/催眠药。

象形图

Skull and crossbones

警示用语:

Danger

危险声明

危险分类

Acute Tox. 3 Oral - Eye Irrit. 2 - Skin Irrit. 2

储存分类代码

6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects

WGK

WGK 2

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Eyeshields, Faceshields, Gloves, type P2 (EN 143) respirator cartridges


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分析证书(COA)

Lot/Batch Number

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Jennifer Ngolab et al.
Acta neuropathologica communications, 8(1), 123-123 (2020-08-06)
An amendment to this paper has been published and can be accessed via the original article.
Xin Fang et al.
Journal of neurochemistry, 150(4), 441-452 (2019-03-10)
While glucagon-like peptide-1 (GLP-1) was reported to have a positive impact on Parkinson disease, it is extremely short half-life greatly hindered its clinical use. In this study, the mouse strain MG1363-pMG36e-GLP-1 was engineered to continuously express GLP-1 to treat Parkinson
Megan M Monsanto et al.
Nature communications, 11(1), 3955-3955 (2020-08-10)
Cellular therapy to treat heart failure is an ongoing focus of intense research, but progress toward structural and functional recovery remains modest. Engineered augmentation of established cellular effectors overcomes impediments to enhance reparative activity. Such 'next generation' implementation includes delivery
Ya-Chen Ko et al.
Journal of anatomy, 214(1), 183-195 (2009-01-27)
CD200 is a highly glycosylated cell surface protein containing two immunoglobulin superfamily domains in the extracellular region and performs immunosuppressive activities. It is widely distributed in various tissues including the vascular endothelium. We report here the distribution of CD200 in
Ya-Fen Jiang-Shieh et al.
Journal of anatomy, 216(3), 407-416 (2010-01-15)
In vivo and in vitro studies have clearly demonstrated that signaling mediated by the interaction of CD200 and its cognate receptor, CD200R, results in an attenuation of inflammatory or autoimmune responses through multiple mechanisms. The present results have shown a

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