所有图片(2)
别名:
2-[p-(2,2-Dichlorocyclopropyl)phenoxy]-2-methylpropanoic acid
经验公式（希尔记法）:
C13H14Cl2O3
CAS号:
分子量:
289.15
EC 号:
MDL编号:
UNSPSC代码:
41116107
PubChem化学物质编号:
NACRES:
NA.77
推荐产品
形式
powder
质量水平
储存温度
2-8°C
SMILES字符串
CC(C)(Oc1ccc(cc1)C2CC2(Cl)Cl)C(O)=O
InChI
1S/C13H14Cl2O3/c1-12(2,11(16)17)18-9-5-3-8(4-6-9)10-7-13(10,14)15/h3-6,10H,7H2,1-2H3,(H,16,17)
InChI key
KPSRODZRAIWAKH-UHFFFAOYSA-N
基因信息
human ... PPARA(5465)
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生化/生理作用
Peroxisome proliferator-activated receptor α (PPARα) agonist
警示用语：
Danger
危险声明
预防措施声明
危险分类
Carc. 1B
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Eyeshields, Gloves, type P3 (EN 143) respirator cartridges
法规信息
新产品
György Paragh et al.
British journal of clinical pharmacology, 61(6), 694-701 (2006-05-26)
Diabetic dyslipidaemia with decreased high-density lipoprotein-cholesterol (HDL-C) concentration plays a key role in enhanced atherosclerosis. The antioxidant effect of HDL is due to the influence of human paraoxonase 1 (PON1) and several authors have described decreased activity of this enzyme
Manfredi Rizzo et al.
Current medical research and opinion, 23(5), 1103-1111 (2007-05-24)
Beyond total low-density-lipoproteins (LDL) levels, increasing evidence suggests that the 'quality' of LDL exerts a great influence on the cardiovascular risk. Several studies have also shown that the therapeutic modulation of LDL size is of benefit in reducing the risk
Zakaria El Kebbaj et al.
Endocrinology, 150(3), 1192-1201 (2008-10-25)
Seasonal obesity and fasting-associated hibernation are the two major metabolic events governing hepatic lipid metabolism in hibernating mammals. In this process, however, the role of the nuclear receptor known as peroxisome proliferator-activated receptor (PPAR)-alpha has not been elucidated yet. Here
Fatemeh Sharifpanah et al.
Stem cells (Dayton, Ohio), 26(1), 64-71 (2007-10-24)
Peroxisome proliferator-activated receptors (PPARalpha, -beta and -gamma) are nuclear receptors involved in transcriptional regulation of lipid and energy metabolism. Since the energy demand increases when cardiac progenitor cells are developing rhythmic contractile activity, PPAR activation may play a critical role
Sung Hun Kim et al.
Toxicology and applied pharmacology, 215(2), 198-207 (2006-04-18)
The present study was undertaken to determine the role of the mitogen-activated protein kinase (MAPK) subfamilies in cell death induced by PPARgamma agonists in osteoblastic cells. Ciglitazone and troglitazone, PPARgamma agonists, resulted in a concentration- and time-dependent cell death, which
Chromatograms
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