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一般描述
m-氨基苯基硼酸-琼脂糖可作为硼酸亲和基质，用于纯化严重急性呼吸综合症(SARS-CoV)核蛋白(NP)抗原。
应用
m-氨基苯基硼酸-琼脂糖已作为分离凝胶，用于对白蛋白进行硼酸亲和层析。它也被用作纯化树脂，用于纯化3,4-二羟基苯乙醛(DOPAL)结合α-突触核蛋白(α-syn)。
外形
悬浮于0.5 M NaCl、0.1 M醋酸钠，pH 5.0。
储存分类代码
10 - Combustible liquids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Serge Chesne et al.
Biochimie, 88(10), 1467-1477 (2006-07-04)
Non-enzymatic glycosylation (glycation) and oxidative damages represent major research areas insofar as such modifications of proteins are frequently observed in numerous states of disease. Albumin undergoes structural and functional alterations, caused by increased glycosylation during non insulin-dependent diabetes mellitus, which
Philippe Rondeau et al.
Archives of biochemistry and biophysics, 460(1), 141-150 (2007-02-27)
Structural modifications of bovine serum albumin (BSA) induced by heating, and the involvement of glycation of albumin in such processing were studied by using Fourier transform infrared spectroscopy (FTIR) and polyacrylamide gel electrophoresis (PAGE). For native BSA, heating treatments gave
N Plotegher et al.
Scientific reports, 7, 40699-40699 (2017-01-14)
Parkinson's disease is a neurodegenerative disorder characterized by the death of dopaminergic neurons and by accumulation of alpha-synuclein (aS) aggregates in the surviving neurons. The dopamine catabolite 3,4-dihydroxyphenylacetaldehyde (DOPAL) is a highly reactive and toxic molecule that leads to aS
Pravin K Bhatnagar et al.
Journal of chromatography. B, Analytical technologies in the biomedical and life sciences, 863(2), 235-241 (2008-02-09)
Hybrid hybridomas (quadromas) are derived by fusing at least two hybridomas, each producing a different antibody of predefined specificity. The resulting cell secretes not only the immunoglobulins of both parents but also hybrid molecules manifesting the binding characteristics of the
Francesco Longo et al.
Acta neuropathologica communications, 5(1), 22-22 (2017-03-16)
Mutations in the leucine-rich repeat kinase 2 (LRRK2) gene are the most common genetic cause of Parkinson's disease. Here, we investigated whether the G2019S LRRK2 mutation causes morphological and/or functional changes at nigro-striatal dopamine neurons. Density of striatal dopaminergic terminals
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