一般描述
抗 c-Myc 过氧化物酶结合物兔抗体与 c-Myc 标记的融合蛋白反应。识别在氨基端或羧基端表达的 c-Myc 标签序列。
特异性
c-Myc (Glu-Gln-Lys-Leu-Ile-Ser-Glu-Glu-Asp-Leu)标记融合蛋白N末端或C末端。
免疫原
人c-myc原致肿瘤基因的肽链对应氨基酸408-425，通过C-末端加成半胱氨酸残基和马来酰亚胺-激活KLH偶联。
外形
溶于 0.01 M 磷酸钠缓冲液 (pH 7.4) 中，含 0.05% MIT汞。
制备说明
用两步法戊二醛法制备（Avrameas, S. 等人，Scand. J. Immunol., 8, Suppl. 7, 7 (1978)）。
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警示用语：
Warning
危险声明
危险分类
Skin Sens. 1
WGK
WGK 2
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
常规特殊物品
Yang Lu et al.
Molecular biology of the cell, 19(10), 4260-4272 (2008-08-08)
Efg1 is essential for hyphal development and virulence in the human pathogenic fungus Candida albicans. How Efg1 regulates gene expression is unknown. Here, we show that Efg1 interacts with components of the nucleosome acetyltransferase of H4 (NuA4) histone acetyltransferase (HAT)
Du-Hwa Lee et al.
The Plant cell, 29(11), 2895-2920 (2017-11-01)
Dynamic control of protein translation in response to the environment is essential for the survival of plant cells. Target of rapamycin (TOR) coordinates protein synthesis with cellular energy/nutrient availability through transcriptional modulation and phosphorylation of the translation machinery. However, mechanisms
Gang Li et al.
Nature cell biology, 13(5), 616-622 (2011-04-19)
The circadian clock controls many metabolic, developmental and physiological processes in a time-of-day-specific manner in both plants and animals. The photoreceptors involved in the perception of light and entrainment of the circadian clock have been well characterized in plants. However
Xiang He et al.
PLoS pathogens, 13(6), e1006347-e1006347 (2017-06-08)
Estrogen-related receptor α (ERRα) is a member of the nuclear receptor superfamily controlling energy homeostasis; however, its precise role in regulating antiviral innate immunity remains to be clarified. Here, we showed that ERRα deficiency conferred resistance to viral infection both
Camilla Kreßner et al.
PloS one, 8(8), e69947-e69947 (2013-08-13)
We have recently characterized SCAI (Suppressor of Cancer Cell Invasion), a transcriptional modulator regulating cancer cell motility through suppression of MAL/SRF dependent gene transcription. We show here that SCAI is expressed in a wide range of normal human tissues and
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