A2562
Angiotensinogen 来源于人类血浆
≥80% (SDS-PAGE)
别名:
Hypertensinogen, Renin substrate
Analysis Note
Protein determined by A1%/280
Packaging
Package size based on protein content.
Biochem/physiol Actions
Angiotensinogen (renin substrate), a component of the rennin-angiotensin system, is a precursor protein to a family of blood pressure and hormone release regulating peptides known as the angiotensins.
Disclaimer
RESEARCH USE ONLY. This product is regulated in France when intended to be used for scientific purposes, including for import and export activities (Article L 1211-1 paragraph 2 of the Public Health Code). The purchaser (i.e. enduser) is required to obtain an import authorization from the France Ministry of Research referred in the Article L1245-5-1 II. of Public Health Code. By ordering this product, you are confirming that you have obtained the proper import authorization.
Physical form
Lyophilized powder containing sodium phosphate buffer, pH 7.
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
高风险级别生物产品--人源产品
此项目有
The renal angiotensin system does not reveal its secrets with a cross-sectional analysis of urinary angiotensinogen levels.
Branko Braam
Journal of hypertension, 31(4), 840-840 (2013-03-08)
Weijian Shao et al.
American journal of physiology. Renal physiology, 304(5), F505-F514 (2013-01-11)
In angiotensin II (ANG II) infusion hypertension, there is an augmentation of intratubular angiotensinogen (AGT) and ANG II leading to increased urinary AGT and ANG II excretion rates associated with tissue injury. However, the changes in urinary AGT and ANG
D A Tewksbury
Federation proceedings, 42(10), 2724-2728 (1983-07-01)
Human angiotensinogen has been purified from outdated blood bank plasma. The purified angiotensinogen has a specific angiotensin I (Ang I) content of 20.7 micrograms Ang I/mg protein and contains only one amino-terminal amino acid, aspartic acid. However, it exhibits two
Angiotensinogen gene polymorphism and HDL2 are linked to coronary artery calcification in individuals with family history of early coronary disease.
Andrei C Sposito et al.
Atherosclerosis, 226(2), 339-340 (2013-01-01)
Jie Yang et al.
Journal of molecular endocrinology, 50(2), R53-R66 (2013-02-02)
The compensatory alterations in the rennin-angiotensin-aldosterone system (RAAS) contribute to the salt-water balance and sufficient placental perfusion for the subsequent well-being of the mother and fetus during normal pregnancy and is characterized by an increase in almost all the components
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