推荐产品
等级
analytical standard, for drug analysis
技术
HPLC: suitable
gas chromatography (GC): suitable
溶解性
H2O: 3.4 mg/mL
ethanol: 5.6 mg/mL
应用
forensics and toxicology
pharmaceutical (small molecule)
格式
neat
SMILES字符串
Cl.CN1CCN2C(C1)c3ccccc3Cc4ccccc24
InChI
1S/C18H20N2.ClH/c1-19-10-11-20-17-9-5-3-7-15(17)12-14-6-2-4-8-16(14)18(20)13-19;/h2-9,18H,10-13H2,1H3;1H
InChI key
YNPFMWCWRVTGKJ-UHFFFAOYSA-N
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应用
Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.
生化/生理作用
Antidepressant; antagonist/inverse agonist at 5-HT2 serotonin receptors; also blocks the H1 histamine receptor and the α2 adrenoceptor.
Yun Gu et al.
Frontiers in pharmacology, 9, 1026-1026 (2018-10-26)
Remyelination is critical for nerve regeneration. However, the molecular mechanism involved in remyelination is poorly understood. To explore the roles of 17β-estradiol (E2) for myelination in the peripheral nervous system, we used a co-culture model of rat dorsal root ganglion
A C de Luca et al.
Journal of tissue engineering and regenerative medicine, 12(3), 676-686 (2017-05-31)
Adipose-derived stem cells (ASC) are becoming one of the most exploited cells in peripheral nerve repair. They are fast-growing and able to protect neurons from apoptosis; they can reduce post-injury latency and the risk of muscle atrophy. This study evaluates
Marike van Lingen et al.
Frontiers in cell and developmental biology, 7, 65-65 (2019-05-21)
Serotonin (5-HT) is a crucial signal in the neurogenic niche microenvironment. Dysregulation of the 5-HT system leads to mood disorders but also to changes in appetite and metabolic rate. Tryptophan hydroxylase 2-deficient (Tph2-/- ) mice depleted of brain 5-HT display
Choongjin Ban et al.
Food chemistry, 302, 125328-125328 (2019-08-14)
To control the oral bioavailability of curcumin, we fabricated solid lipid nanoparticles (SLNs) using tristearin and polyethylene glycol (PEG)ylated emulsifiers. Lipolysis of prepared SLNs via simulated gastrointestinal digestion was modulated by altering the types and concentrations of emulsifiers. After digestion
Boaz Barak et al.
Nature neuroscience, 22(5), 700-708 (2019-04-24)
Williams syndrome (WS), caused by a heterozygous microdeletion on chromosome 7q11.23, is a neurodevelopmental disorder characterized by hypersociability and neurocognitive abnormalities. Of the deleted genes, general transcription factor IIi (Gtf2i) has been linked to hypersociability in WS, although the underlying
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