SMILES string
[S](=O)(=O)(O)O.N(C(C)C)CC(O)c2cc(c(cc2)O)O.N(C(C)C)CC(O)c1cc(c(cc1)O)O.O.O
InChI
1S/2C11H17NO3.H2O4S.2H2O/c2*1-7(2)12-6-11(15)8-3-4-9(13)10(14)5-8;1-5(2,3)4;;/h2*3-5,7,11-15H,6H2,1-2H3;(H2,1,2,3,4);2*1H2
InChI key
CUQPTVCVZLUXJB-UHFFFAOYSA-N
grade
pharmaceutical primary standard
API family
isoprenaline
manufacturer/tradename
EDQM
application(s)
pharmaceutical (small molecule)
format
neat
storage temp.
2-8°C
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
Yafei Zhang et al.
Nan fang yi ke da xue xue bao = Journal of Southern Medical University, 33(10), 1517-1520 (2013-10-23)
To observe the changes of surface ECG and cell couplings between sinoatrial node cells and myocardial cells following transplantion of pedicled autologous sinoatrial node tissue graft into the right ventricle of a canine model of complete atrioventricular block. Ten healthy
Keyvan Yousefi et al.
Indian journal of experimental biology, 51(8), 653-660 (2013-11-16)
Isoproterenol injection (100 mg/kg; sc) produced changes in ECG pattern including ST-segment elevation and suppressed R-amplitude. The methanolic extract of M. vulgare at doses of 10, 20, and 40 mg/kg significantly amended the ECG changes. A severe myocardial necrosis and
George King et al.
American journal of physiology. Lung cellular and molecular physiology, 306(3), L292-L298 (2013-12-10)
Glucocorticoids induce lung fibroblasts to produce fibroblast-pneumocyte factor, a peptide that stimulates type II cells to synthesize pulmonary surfactant. This effect is known to be more apparent in cells derived from female fetuses, a characteristic that has been attributed to
Masanobu Kawai et al.
The Journal of biological chemistry, 289(3), 1457-1466 (2013-12-05)
The circadian clock network is well known to link food intake and metabolic outputs. Phosphorus is a pivotal nutritional factor involved in energy and skeletal metabolisms and possesses a circadian profile in the circulation; however, the precise mechanisms whereby phosphate
Birgül Kurt et al.
American journal of physiology. Renal physiology, 306(3), F327-F332 (2013-11-29)
In states of loss-of-function mutations of the renin-angiotensin-aldosterone system, kidneys develop a strong hyperplasia of renin-producing cells. Those additional renin cells are located outside the classic juxtaglomerular areas, mainly in the walls of preglomerular vessels and most prominently in multilayers
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