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Merck
CN

BP453

硝普钠 二水合物

British Pharmacopoeia (BP) Reference Standard

别名:

SNP, 五氰基亚硝酰基铁酸钠, 硝基铁酸氰化钠, 硝普盐 钠

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About This Item

线性分子式:
Na2[Fe(CN)5NO] · 2H2O
CAS号:
分子量:
297.95
EC 号:
MDL编号:
UNSPSC代码:
41116107
NACRES:
NA.24

等级

pharmaceutical primary standard

API类

sodium nitroprusside

形式

solid

保质期

limited shelf life, expiry date on the label

制造商/商品名称

BP

应用

pharmaceutical
pharmaceutical small molecule

格式

neat

储存温度

2-8°C

SMILES字符串

[Na+].[Na+].[H]O[H].[H]O[H].O=N[Fe--](C#N)(C#N)(C#N)(C#N)C#N

InChI

1S/5CN.Fe.NO.2Na.2H2O/c5*1-2;;1-2;;;;/h;;;;;;;;;2*1H2/q;;;;;2*-1;2*+1;;

InChI key

OIRZWVYIQXBRFC-UHFFFAOYSA-N

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一般描述

This product is provided as delivered and specified by the issuing Pharmacopoeia. All information provided in support of this product, including SDS and any product information leaflets have been developed and issued under the Authority of the issuing Pharmacopoeia.For further information and support please go to the website of the issuing Pharmacopoeia.

应用

Sodium nitroprusside BP Reference standard, intended for use in laboratory tests only as specifically prescribed in the British Pharmacopoeia.

Also used in monographs such as:
  • Sodium Nitroprusside Infusion
  • 包装

    Unit quantity: 200 mg. Subject to change. The product is delivered as supplied by the issuing Pharmacopoeia. For the current unit quantity please visit British Pharmacopoeia

    其他说明

    Sales restrictions may apply.

    象形图

    Skull and crossbones

    警示用语:

    Danger

    危险声明

    预防措施声明

    危险分类

    Acute Tox. 3 Oral

    WGK

    WGK 3

    闪点(°F)

    Not applicable

    闪点(°C)

    Not applicable


    Choose from one of the most recent versions:

    分析证书(COA)

    Lot/Batch Number

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    Bin He et al.
    BMC complementary medicine and therapies, 20(1), 23-23 (2020-02-06)
    Chondrocyte apoptosis activated by the mitochondrial dependent pathway serves a crucial role in cartilage degeneration of osteoarthritis (OA). In the present study, the protective effects of CMCS against sodium nitroprusside (SNP)-induced chondrocyte apoptosis were evaluated and the underlying molecular mechanisms

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