产品线
BioXtra
质量水平
检测方案
≥98.0%
形式
powder
杂质
≤0.0005% Phosphorus (P)
≤0.005% Insoluble matter
mp
92 °C (lit.)
溶解性
H2O: 0.5 M, clear, colorless
密度
1.757 g/mL at 25 °C (lit.)
痕量阴离子
chloride (Cl-): ≤0.0005%
痕量阳离子
Ca: ≤0.0005%
Cu: ≤0.0005%
Fe: ≤0.001%
Mg: ≤0.005%
NH4+: ≤0.005%
Na: ≤0.02%
Pb: ≤0.001%
Zn: ≤0.0005%
SMILES字符串
O.O.O.O.O.O.O.O.O.O.O.O.[Al+3].[K+].[O-]S([O-])(=O)=O.[O-]S([O-])(=O)=O
InChI
1S/Al.K.2H2O4S.12H2O/c;;2*1-5(2,3)4;;;;;;;;;;;;/h;;2*(H2,1,2,3,4);12*1H2/q+3;+1;;;;;;;;;;;;;;/p-4
InChI key
GNHOJBNSNUXZQA-UHFFFAOYSA-J
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WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Eyeshields, Gloves, type N95 (US)
Nature communications, 5, 4273-4273 (2014-07-01)
Germline or B-cell-specific loss of Ptpn6 gene encoding the Shp1 protein tyrosine phosphatase leads to skewed B lymphopoiesis and systemic autoimmunity. Here, to study its role in B-cell terminal differentiation, we generated Ptpn6(f/f)Aicda(Cre/+) mice with Shp1 ablated only in activated
The Journal of experimental medicine, 211(6), 1019-1025 (2014-05-07)
There is currently no paradigm in immunology that enables an accurate prediction of how the immune system will respond to any given agent. Here we show that the immunological responses induced by members of a broad class of inorganic crystalline
The Journal of experimental medicine, 211(7), 1297-1305 (2014-06-11)
Follicular helper T (Tfh) cells access the B cell follicle to promote antibody responses and are particularly important for germinal center (GC) reactions. However, the molecular mechanisms of how Tfh cells are physically associated with GCs are incompletely understood. We
Advanced healthcare materials, 3(11), 1898-1908 (2014-06-14)
Epitope content plays a critical role in determining T-cell and antibody responses to vaccines, biomaterials, and protein therapeutics, but its effects are nonlinear and difficult to isolate. Here, molecular self-assembly is used to build a vaccine with precise control over
American journal of rhinology & allergy, 28(4), 290-296 (2014-09-10)
The naso-ocular interaction in allergic rhinoconjunctivitis is well recognized from epidemiological, clinical, and experimental observations. The precise mechanisms remain incompletely understood. A new mouse model of allergic rhinoconjunctivitis was used to investigate the contribution of mast cells and trigeminal ganglia
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