所有图片(1)
About This Item
经验公式（希尔记法）:
C6H3N3O9S
CAS号:
分子量:
293.17
Beilstein:
572358
MDL编号:
UNSPSC代码:
12352005
PubChem化学物质编号:
NACRES:
NA.25
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应用
亲水改性剂。 具有氨基化合物的衍生物可以通过肼进行再生。
苦味磺酸（2,4,6-三硝基苯磺酸溶液，TNBS）可用于研究细胞内pH的调节机制。 TNBS可用于诱发炎症性肠病（IBD）/结肠炎，从而用于其机理和治疗方法的研究。
注意
在≥0°C下保存时会发生降解，并形成沉淀（苦味酸）
其他说明
销售限制可能适用
警示用语：
Danger
危险分类
Acute Tox. 4 Oral - Desen. Expl. 3 - Eye Dam. 1 - Skin Corr. 1B - Skin Sens. 1
储存分类代码
3 - Flammable liquids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter
法规信息
监管及禁止进口产品
Ana Beatriz Albino de Almeida et al.
Journal of ethnopharmacology, 146(1), 300-310 (2013-01-15)
In Brazilian traditional medicine, Arctium lappa (Asteraceae), has been reported to relieve gastrointestinal symptoms. In the present study, we investigated the effects of the lactone sesquiterpene onopordopicrin enriched fraction (ONP fraction) from Arctium lappa in an experimental colitis model induced
Joel Castro et al.
Gastroenterology, 145(6), 1334-1346 (2013-08-21)
Linaclotide is a minimally absorbed agonist of guanylate cyclase-C (GUCY2C or GC-C) that reduces symptoms associated with irritable bowel syndrome with constipation (IBS-C). Little is known about the mechanism by which linaclotide reduces abdominal pain in patients with IBS-C. We
Øystein Brenna et al.
PloS one, 8(1), e54543-e54543 (2013-02-06)
Rectal instillation of trinitrobenzene sulphonic acid (TNBS) in ethanol is an established model for inflammatory bowel disease (IBD). We aimed to 1) set up a TNBS-colitis protocol resulting in an endoscopic and histologic picture resembling IBD, 2) study the correlation
Bas Kremer et al.
PloS one, 7(11), e50388-e50388 (2012-12-12)
The recurrent TNBS-colitis model in BALB/c mice has been proposed as a model of Inflammatory Bowel Disease with a shifting pattern of local cytokines with the expression of Th1 cytokines during the early phase, Th17 cytokines during the intermediate phase
Weicheng Liu et al.
The Journal of clinical investigation, 123(9), 3983-3996 (2013-08-16)
The inhibitory effects of vitamin D on colitis have been previously documented. Global vitamin D receptor (VDR) deletion exaggerates colitis, but the relative anticolitic contribution of epithelial and nonepithelial VDR signaling is unknown. Here, we showed that colonic epithelial VDR
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