80827
N,N-Dimethyl-2-sulfamoylnicotinamide
PESTANAL®, analytical standard
别名:
2-(Aminosulfonyl)-N,N-dimethyl-3-pyridinecarboxamide, 2-Aminosulfonyl-N,N-dimethylnicotinamide, N,N-Dimethyl-2-sulfamoylpyridine-3-carboxamide
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所有图片(1)
About This Item
经验公式（希尔记法）:
C8H11N3O3S
CAS号:
分子量:
229.26
Beilstein:
9193761
MDL编号:
UNSPSC代码:
12352200
PubChem化学物质编号:
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等级
analytical standard
产品线
PESTANAL®
方案
≥98.0% (HPLC)
保质期
limited shelf life, expiry date on the label
包装形式
neat
储存温度
2-8°C
SMILES字符串
O=C(N(C)C)C1=CC=CN=C1S(=O)(N)=O
InChI
1S/C8H11N3O3S/c1-11(2)8(12)6-4-3-5-10-7(6)15(9,13)14/h3-5H,1-2H3,(H2,9,13,14)
InChI key
WYFKZPLSYVJLRB-UHFFFAOYSA-N
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Find a digital Reference Material for this product available on our online platform ChemisTwin® for NMR. You can use this digital equivalent on ChemisTwin® for your sample identity confirmation and compound quantification (with digital external standard). An NMR spectrum of this substance can be viewed and an online comparison against your sample can be performed with a few mouseclicks. Learn more here and start your free trial.
法律信息
PESTANAL is a registered trademark of Merck KGaA, Darmstadt, Germany
警示用语：
Warning
危险声明
危险分类
Aquatic Chronic 3 - Skin Sens. 1
储存分类代码
11 - Combustible Solids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
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Nature cell biology, 22(12), 1436-1446 (2020-12-02)
X-chromosome dosage compensation in female placental mammals is achieved by X-chromosome inactivation (XCI). Human pre-implantation embryos are an exception, in which dosage compensation occurs by X-chromosome dampening (XCD). Here, we examined whether XCD extends to human prenatal germ cells given
Ting Gang Chew et al.
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Eukaryotic cells assemble actomyosin rings during cytokinesis to function as force-generating machines to drive membrane invagination and to counteract the intracellular pressure and the cell surface tension. How the extracellular matrix affects actomyosin ring contraction has not been fully explored.
Mette C Jørgensen et al.
Development (Cambridge, England), 145(17) (2018-08-11)
Mutations in Hes1, a target gene of the Notch signalling pathway, lead to ectopic pancreas by a poorly described mechanism. Here, we use genetic inactivation of Hes1 combined with lineage tracing and live imaging to reveal an endodermal requirement for
Vladislav Belyy et al.
Proceedings of the National Academy of Sciences of the United States of America, 117(3), 1533-1542 (2019-12-25)
The endoplasmic reticulum (ER) membrane-resident stress sensor inositol-requiring enzyme 1 (IRE1) governs the most evolutionarily conserved branch of the unfolded protein response. Upon sensing an accumulation of unfolded proteins in the ER lumen, IRE1 activates its cytoplasmic kinase and ribonuclease
María Expósito-Serrano et al.
Current biology : CB, 30(16), 3212-3222 (2020-06-06)
An important question in cell biology is how cellular organelles partition during cell division. In organisms undergoing closed mitosis, the elongation of an intranuclear spindle drives nuclear division, generating two identically sized nuclei [1, 2]. However, how the site of
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