Agency
USP/NF
tested according to Ph. Eur.
质量水平
蒸汽压
1.33 (1.00 mmHg) at 865 °C hPa
检测方案
99.7% (dried substance)
形式
solid
pH值（酸碱度）
7
mp
801 °C (lit.)
溶解性
water: soluble (358 g/l at 20 °C (68 °F))
SMILES字符串
[Na+].[Cl-]
InChI
1S/ClH.Na/h1H;/q;+1/p-1
InChI key
FAPWRFPIFSIZLT-UHFFFAOYSA-M
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WGK
WGK 1
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Eyeshields, Gloves, type N95 (US)
Francis Kim et al.
JAMA, 311(1), 45-52 (2013-11-19)
Hospital cooling improves outcome after cardiac arrest, but prehospital cooling immediately after return of spontaneous circulation may result in better outcomes. To determine whether prehospital cooling improves outcomes after resuscitation from cardiac arrest in patients with ventricular fibrillation (VF) and
Alexander W Wyatt et al.
Genome biology, 15(8), 426-426 (2014-08-27)
Genomic analyses of hundreds of prostate tumors have defined a diverse landscape of mutations and genome rearrangements, but the transcriptomic effect of this complexity is less well understood, particularly at the individual tumor level. We selected a cohort of 25
Koji Tanaka et al.
Nature communications, 6, 6337-6337 (2015-02-27)
Pore-forming toxins (PFT) are water-soluble proteins that possess the remarkable ability to self-assemble on the membrane of target cells, where they form pores causing cell damage. Here, we elucidate the mechanism of action of the haemolytic protein fragaceatoxin C (FraC)
Frédéric Lebrun-Julien et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 34(25), 8432-8448 (2014-06-21)
The mammalian target of rapamycin (mTOR) pathway integrates multiple signals and regulates crucial cell functions via the molecular complexes mTORC1 and mTORC2. These complexes are functionally dependent on their raptor (mTORC1) or rictor (mTORC2) subunits. mTOR has been associated with
Gemma L Carvill et al.
Neurology, 82(14), 1245-1253 (2014-03-14)
To determine the genes underlying Dravet syndrome in patients who do not have an SCN1A mutation on routine testing. We performed whole-exome sequencing in 13 SCN1A-negative patients with Dravet syndrome and targeted resequencing in 67 additional patients to identify new
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