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安全信息

18304

Sigma-Aldrich

过氧化氢 溶液

34.5-36.5%

别名:

H2O2, Hydrogen Peroxide (H2O2)

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5 L
¥4,036.69

¥4,036.69


预计发货时间2025年7月01日详情


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5 L
¥4,036.69

About This Item

经验公式(希尔记法):
H2O2
CAS号:
分子量:
34.01
Beilstein:
3587191
MDL编号:
UNSPSC代码:
12352300
PubChem化学物质编号:
NACRES:
NA.21

¥4,036.69


预计发货时间2025年7月01日详情


获取大包装报价

蒸汽压

23.3 mmHg ( 30 °C)

质量水平

方案

34.5-36.5%

保质期

Shelf life is limited.

包含

stabilizer

反应适用性

reagent type: oxidant

杂质

≤0.0002% heavy metals (as Pb)
≤0.02% free acid (as H2SO4)
≤0.05% non-volatile matter

灼烧残渣

≤0.03%

溶解性

diethyl ether: soluble
petroleum ether: insoluble
water: soluble

密度

1.130 g/cm3

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此商品
13191013192095299
assay

34.5-36.5%

assay

-

assay

-

assay

≥35% (RT)

Quality Level

200

Quality Level

-

Quality Level

-

Quality Level

100

solubility

diethyl ether: soluble, water: soluble, petroleum ether: insoluble

solubility

-

solubility

-

solubility

-

reaction suitability

reagent type: oxidant

reaction suitability

reagent type: oxidant

reaction suitability

reagent type: oxidant

reaction suitability

reagent type: oxidant

density

1.130 g/cm3

density

1.13 g/cm3

density

1.13 g/cm3

density

1.13 g/mL at 20 °C

storage temp.

2-8°C

storage temp.

-

storage temp.

-

storage temp.

2-8°C

应用

过氧化氢已用于 eNOS 的免疫细胞化学。,[1]还用于抗凝血酶原和抗 β2-糖蛋白 I 抗体检测显色。[2]此外,它还用作Hanks缓冲盐溶液补充剂,用于评估髓过氧化物酶的功能活性。[3]

生化/生理作用

过氧化氢 (H2O2)是一种强氧化剂,可通过凋亡途径诱导培养的皮质神经元中的神经元死亡。[4]过氧化氢促进的氧化爆发(oxidative burst)是不相容相互作用过程中植物防御的关键机制。注入转基因水稻中的过氧化氢可诱导细胞死亡,并增加对细菌和真菌病原体的抵抗力。[5]

注意

在黑暗处保存

其他说明

货号18304-4X5L将停产。请订购具有相同物理性质和规格的单瓶18304-5L。
货号18304-6X1L的产品即将停产。请订购18304-1L单瓶装,因为其物理参数与上述产品本质相同。

象形图

CorrosionExclamation mark

警示用语:

Danger

危险分类

Acute Tox. 4 Oral - Aquatic Chronic 3 - Eye Dam. 1 - Skin Irrit. 2 - STOT SE 3

靶器官

Respiratory system

储存分类代码

5.1B - Oxidizing hazardous materials

WGK

WGK 1

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter

法规信息

危险化学品
易制爆化学品
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    Aardra Kachroo et al.
    Transgenic research, 12(5), 577-586 (2003-11-07)
    Oxidative burst, mediated by hydrogen peroxide (H2O2), has been recognized as a key component of plant defense response during an incompatible interaction. To determine if elevated levels of H2O2 lead to cell death, activation of defense genes and enhanced resistance
    F J Muñoz-Rodríguez et al.
    Haematologica, 85(6), 632-637 (2000-06-28)
    Antibodies to prothrombin (aII) have been identified in patients with antiphospholipid antibodies, but their clinical significance is not well known. The aim of our study was to investigate their prevalence and association with clinical manifestations of the antiphospholipid syndrome (APS)
    E R Whittemore et al.
    Neuroscience, 67(4), 921-932 (1995-08-01)
    A variety of neurodegenerative disease states have been associated with oxidative damage or stress. Such stress is thought to be mediated by excessive exposure of cells to reactive oxygen species such as free radicals, which can be generated following cell
    J F Telfer et al.
    Molecular human reproduction, 3(1), 69-75 (1997-01-01)
    Immunocytochemistry was used to localize endothelial (eNOS) and inducible (iNOS) nitric oxide synthase in human uterine tissues collected at various stages of the menstrual cycle, after exposure to exogenous progestagens, and in early pregnancy. Endothelial NOS-like immunoreactivity was detected in
    R J Falk et al.
    Clinical and experimental immunology, 89(2), 274-278 (1992-08-01)
    We wondered whether anti-myeloperoxidase (MPO) autoantibodies (MPO-ANCA) found in patients with systemic vasculitis react with a conformational epitope or epitopes on the MPO molecule. Sera from 15 human MPO-ANCA, and a polyclonal and a monoclonal anti-MPO antibodies were reacted with

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