18-1630
氯化锂 溶液
suitable for atomic absorption spectrometry, 1 mg/mL Li, 1000 ppm Li
别名:
Lithium chloride solution
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关于此项目
线性分子式:
LiCl
化学文摘社编号:
分子量:
42.39
UNSPSC Code:
12352302
PubChem Substance ID:
MDL number:
Grade:
SAJ special grade
Form:
liquid
InChI
1S/ClH.Li/h1H;/q;+1/p-1
SMILES string
[Li+].[Cl-]
InChI key
KWGKDLIKAYFUFQ-UHFFFAOYSA-M
grade
SAJ special grade
form
liquid
availability
available only in Japan
concentration
1 mg/mL Li, 1000 ppm Li
suitability
suitable for atomic absorption spectrometry
storage temp.
15-25°C
signalword
Warning
hcodes
pcodes
Hazard Classifications
Met. Corr. 1
flash_point_f
Not applicable
flash_point_c
Not applicable
存储类别
12 - Non Combustible Liquids
法规信息
新产品
此项目有
Otilia Zarnescu et al.
Micron (Oxford, England : 1993), 46, 66-75 (2013-01-29)
In the present study we examined the effects of lithium chloride on the Corydoras aeneus caudal fin regeneration. After caudal fin amputation, the fish were exposed 3h daily to 35 mM lithium chloride for 9 days. The effects of lithium
Li-Wei Zheng et al.
International journal of oral science, 5(1), 1-6 (2013-03-30)
This study aimed to compare epithelial cells derived from human embryonic stem cells (hESCs) to human ameloblast-lineage cells (ALCs), as a way to determine their potential use as a cell source for ameloblast regeneration. Induced by various concentrations of bone
Aitor González et al.
PloS one, 8(1), e53323-e53323 (2013-01-18)
The mouse segmentation is established from somites, which are iteratively induced every two hours from the presomitic mesoderm (PSM) by a system known as the segmentation clock. A crucial component of the segmentation clock is the gene Hes7, which is
Weiwei Gao et al.
Carcinogenesis, 34(6), 1208-1215 (2013-02-08)
We recently reported that an N-terminally truncated retinoid X receptor-α (tRXRα) produced in cancer cells acts to promote cancer cell growth and survival through AKT activation. However, how RXRα is cleaved and how the cleavage is regulated in cancer cells
A Briolay et al.
Biochemical and biophysical research communications, 430(3), 1072-1077 (2012-12-26)
Although anti-tumor necrosis factor (TNF)-α treatments efficiently block inflammation in ankylosing spondylitis (AS), they are inefficient to prevent excessive bone formation. In AS, ossification seems more prone to develop in sites where inflammation has resolved following anti-TNF therapy, suggesting that
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