13-1890
氢氟酸
55%, CP
别名:
HF
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关于此项目
经验公式（希尔记法）:
HF
化学文摘社编号:
分子量:
20.01
UNSPSC Code:
12352106
PubChem Substance ID:
MDL number:
Concentration:
55%
Form:
liquid
Grade:
CP
InChI
1S/FH/h1H
SMILES string
[H+].[F-]
grade
CP
vapor pressure
25 mmHg ( 20 °C)
form
liquid
availability
available only in Japan
concentration
55%
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signalword
Danger
hcodes
Hazard Classifications
Acute Tox. 1 Dermal - Acute Tox. 2 Inhalation - Acute Tox. 2 Oral - Eye Dam. 1 - Skin Corr. 1A
存储类别
6.1B - Non-combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials
wgk
WGK 2
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
新产品
此项目有
Ian Ford et al.
International journal of cardiology, 184, 163-169 (2015-02-24)
We identified easily obtained baseline characteristics associated with outcomes in patients with chronic heart failure (HF) and elevated heart rate (HR) receiving contemporary guideline-recommended therapy in the SHIFT trial, and used them to develop a prognostic model. We selected the
Faiez Zannad et al.
European journal of heart failure, 17(7), 735-742 (2015-04-29)
Thrombin is a critical element of crosstalk between pathways contributing to worsening of established heart failure (HF). The aim of this study is to explore the efficacy and safety of rivaroxaban 2.5 mg bid compared with placebo (with standard care) after
Barry Greenberg et al.
JACC. Heart failure, 2(1), 84-92 (2014-03-14)
Impaired cardiac isoform of sarco(endo)plasmic reticulum Ca(2+) ATPase (SERCA2a) activity is a key abnormality in heart failure patients with reduced ejection fraction. The CUPID 2 (Calcium Up-Regulation by Percutaneous Administration of Gene Therapy in Cardiac Disease Phase 2b) trial is
Philip J Millar et al.
Circulation, 131(5), 459-468 (2014-12-04)
Muscle sympathetic activation in heart failure with reduced ejection fraction (HFrEF) has been attributed, on the basis of multiunit recordings, to attenuated inhibitory feedback from stretch-sensitive cardiopulmonary mechanoreceptors. However, such preparations integrate 2 populations of single units exhibiting directionally opposite
Nicolas Vodovar et al.
JACC. Heart failure, 3(8), 629-636 (2015-08-08)
This study sought to hypothesize that elevated B-type natriuretic peptide (BNP) could act as an endogenous neprilysin inhibitor. A hallmark of acute decompensated heart failure (ADHF) is the overproduction of natriuretic peptides (NPs) by stretched cardiomyocytes. Various strategies have been
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