10775835001
Roche
牛血清白蛋白
98.5% (electrophoresis), Fatty acids (total) ≤0.2 mg/g, Triglycerides (enzym.) free, Immunoglobulins not detectable, microbiological culture: suitable, USA origin
别名:
BSA, Bovine Serum Albumin Fraction V, fatty acid free, Bovine albumin, bovine serum
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About This Item
UNSPSC代码:
12352106
推荐产品
生物来源
bovine
质量水平
方案
98.5% (electrophoresis)
表单
lyophilized
分子量
Mr 68 kDa
包装
pkg of 50 g
制造商/商品名称
Roche
来源
USA origin
技术
immunofluorescence: suitable
ligand binding assay: suitable
杂质
Immunoglobulins, none detected
Triglycerides, none detected
≤0.2 mg/g fatty acids
UniProt登记号
运输
wet ice
储存温度
2-8°C
基因信息
bovine ... ALB(280717)
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一般描述
Highest quality for fatty acid and lipid research.
BSA is a single polypeptide chain consisting of about 583 amino acid residues and no carbohydrates. At pH 5-7, it contains 17 intra-chain disulfide bridges and one sulfhydryl group.
BSA is a single polypeptide chain consisting of about 583 amino acid residues and no carbohydrates. At pH 5-7, it contains 17 intra-chain disulfide bridges and one sulfhydryl group.
应用
Bovine Serum Albumin, Fraction V, fatty acid free is used:
- in lipid research to study lipid metabolism in biological systems
- to identify transport mechanisms in living organisms
- to study ligand binding
- to investigate ionic binding
- in the preparation of solutions during isolation of cardiomyocytes
- as a blocking solution to incubate embryos for immunofluorescence
- in dual polarisation interferometry (DPI) to study the interaction between membrane active peptides and artificial bilayers
生化/生理作用
The fatty acids that are insoluble in circulating plasma are transported primarily by serum albumin. The human serum albumin and bovine serum albumin share 76% sequence homology. Being the most abundant protein in the circulatory system, it participates in the maintenance of blood pH and carries several endogenous and exogenous compounds, including fatty acids, metal, amino acids, steroids and drugs.
特点和优势
- Free from fatty acids according to current quality control procedures (suitable for lipid research)
- High quality
- Low content of heavy metal ions
- Long-term stability
质量
Purity: ≥98.5 % albumin (electrophoresis), 98% protein
Contaminants: <0.2 mg fatty acids/g, triglycerides not detectable (enzymatic), immunoglobulins not detectable (Ouchterlony), ≤5% H2O,≤30 ppm heavy metals
Contaminants: <0.2 mg fatty acids/g, triglycerides not detectable (enzymatic), immunoglobulins not detectable (Ouchterlony), ≤5% H2O,≤30 ppm heavy metals
其他说明
仅用于生命科学研究。不可用于诊断。
储存分类代码
11 - Combustible Solids
WGK
WGK 1
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
监管及禁止进口产品
Examination of the Interaction between a Membrane Active Peptide
and Artificial Bilayers by Dual Polarisation Interferometry
and Artificial Bilayers by Dual Polarisation Interferometry
Payne JAE, et al.
Bio-protocol, 7(1), 1-12 (2017)
Relief of endoplasmic reticulum stress enhances DNA damage repair and improves development of pre-implantation embryos
Dicks N, et al.
PLoS ONE, 12(11), e0187717-e0187717 (2017)
Jelena Branovets et al.
American journal of physiology. Heart and circulatory physiology, 305(4), H506-H520 (2013-06-25)
Disruption of the creatine kinase (CK) system in hearts of CK-deficient mice leads to changes in the ultrastructure and regulation of mitochondrial respiration. We expected to see similar changes in creatine-deficient mice, which lack the enzyme guanidinoacetate methyltransferase (GAMT) to
Geronimo Matteo et al.
Endocrinology, 162(6) (2021-03-12)
Human studies consistently show an association between exposure to persistent organic pollutants, including 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD, aka "dioxin"), and increased diabetes risk. We previously showed that a single high-dose TCDD exposure (20 µg/kg) decreased plasma insulin levels in male and female
Tiziana Caputo et al.
Cellular and molecular life sciences : CMLS, 78(1), 227-247 (2020-03-12)
Chronic inflammation that affects primarily metabolic organs, such as white adipose tissue (WAT), is considered as a major cause of human obesity-associated co-morbidities. However, the molecular mechanisms initiating this inflammation in WAT are poorly understood. By combining transcriptomics, ChIP-seq and
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