推荐产品
生物来源
mouse
质量水平
抗体形式
purified immunoglobulin
抗体产品类型
primary antibodies
克隆
OM84, monoclonal
种属反应性
human
制造商/商品名称
Chemicon®
技术
ELISA: suitable
western blot: suitable
同位素/亚型
IgG2aκ
UniProt登记号
运输
dry ice
靶向翻译后修饰
unmodified
基因信息
human ... APP(351)
特异性
Reacts with secretory amyloid precursor protein (sAPP), but not with Amyloid-Beta protein 1-40.
免疫原
Amyloid Precursor Protein (APP) secreted by the EJ-1 bladder carcinoma cell line.
Epitope: Secretory
应用
Anti-Amyloid Precursor Protein Antibody, Secretory, clone OM84 detects level of Amyloid Precursor Protein & has been published & validated for use in ELISA & WB.
Research Category
Neuroscience
Neuroscience
Research Sub Category
Neurodegenerative Diseases
Neurodegenerative Diseases
Western blot: 10 μg/mL ELISA: 1 μg/mL
Optimal working dilutions must be determined by end user.
Optimal working dilutions must be determined by end user.
外形
Protein A purified
Format: Purified
Protein A Purified mouse immunoglobulin in 10 mM sodium phosphate buffer (pH 7.4), 0.15 M NaCl, and 0.1% sodium azide as a preservative..
储存及稳定性
Maintain frozen at -70°C or below in undiluted aliquots for up to 12 months, avoid repeated freeze/thaw cycles.
分析说明
Control
Brain
Brain
其他说明
Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.
法律信息
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
免责声明
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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储存分类代码
12 - Non Combustible Liquids
WGK
WGK 2
闪点(°F)
Not applicable
闪点(°C)
Not applicable
The Journal of neuroscience : the official journal of the Society for Neuroscience, 35(20), 7777-7794 (2015-05-23)
Stroke-induced immunodepression (SIDS) is an essential cause of poststroke infections. Pharmacological inhibition of SIDS appears promising in preventing life-threatening infections in stroke patients. However, SIDS might represent an adaptive mechanism preventing autoreactive immune responses after stroke. To address this, we
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