推荐产品
生物来源
mouse
质量水平
抗体形式
purified immunoglobulin
抗体产品类型
primary antibodies
克隆
8F6.2, monoclonal
种属反应性
monkey, fish, rabbit
制造商/商品名称
Chemicon®
技术
immunohistochemistry: suitable
同位素/亚型
IgG1
UniProt登记号
运输
dry ice
靶向翻译后修饰
unmodified
基因信息
rabbit ... Gjd2(100341036)
特异性
可识别鲈鱼连接蛋白35。不与连接蛋白34.7（一种密切相关的蛋白）发生交叉反应。还识别兔和猕猴连接蛋白36，即连接蛋白35的基因同源物。在免疫组化中标记鲈鱼，兔子和猕猴视网膜中的间隙连接。
应用
使用经验证可用于IH的抗连接蛋白35/36抗体（克隆8F6.2）检测连接蛋白35/36。
联系
替代品：MAB3044
外形
形式：纯化
纯化的免疫球蛋白。 液体溶于含50%甘油的0.1 M柠檬酸盐缓冲液中，还含有0.1%叠氮化钠作为防腐剂
其他说明
浓度：请参考批次特异性浓缩物的分析证书。
法律信息
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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储存分类代码
12 - Non Combustible Liquids
WGK
WGK 2
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Elena Ivanova et al.
Frontiers in cellular neuroscience, 9, 390-390 (2015-10-21)
Retinal degeneration (RD) encompasses a family of diseases that lead to photoreceptor death and visual impairment. Visual decline due to photoreceptor cell loss is further compromised by emerging spontaneous hyperactivity in inner retinal cells. This aberrant activity acts as a
Adam C Miller et al.
eLife, 6 (2017-05-23)
Neural network function is based upon the patterns and types of connections made between neurons. Neuronal synapses are adhesions specialized for communication and they come in two types, chemical and electrical. Communication at chemical synapses occurs via neurotransmitter release whereas
Adenosine and dopamine receptors coregulate photoreceptor coupling via gap junction phosphorylation in mouse retina.
Li, H; Zhang, Z; Blackburn, MR; Wang, SW; Ribelayga, CP; O'Brien, J
The Journal of Neuroscience null
J E Rash et al.
Journal of neurocytology, 33(1), 131-151 (2004-06-03)
Combined confocal microscopy and freeze-fracture replica immunogold labeling (FRIL) were used to examine the connexin identity at electrical synapses in goldfish brain and rat retina, and to test for "co-localization" vs. "close proximity" of connexins to other functionally interacting proteins
Christopher H Emfinger et al.
Physiological reports, 7(11), e14101-e14101 (2019-06-05)
Islet β-cell membrane excitability is a well-established regulator of mammalian insulin secretion, and defects in β-cell excitability are linked to multiple forms of diabetes. Evolutionary conservation of islet excitability in lower organisms is largely unexplored. Here we show that adult
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